p38 and ERK1/2 MAPKs mediate the interplay of TNF-alpha and IL-10 in regulating oxidative stress and cardiac myocyte apoptosis. - Test2 - elhamkhani - TriplyDB

p38 and ERK1/2 MAPKs mediate the interplay of TNF-alpha and IL-10 in regulating oxidative stress and cardiac myocyte apoptosis.

p38 and ERK1/2 MAPKs mediate the interplay of TNF-alpha and IL-10 in regulating oxidative stress and cardiac myocyte apoptosis.

http://www.wikidata.org/entity/Q46974170

about

WISP1, a pro-mitogenic, pro-survival factor, mediates tumor necrosis factor-alpha (TNF-alpha)-stimulated cardiac fibroblast proliferation but inhibits TNF-alpha-induced cardiomyocyte death Reactive oxygen species at the crossroads of inflammasome and inflammation Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.Time course of changes in oxidative stress and stress-induced proteins in cardiomyocytes exposed to doxorubicin and prevention by vitamin C.Akt regulates IL-10 mediated suppression of TNFα-induced cardiomyocyte apoptosis by upregulating Stat3 phosphorylation.Cross-talk between cAMP and MAPK pathways in HSD11B2 induction by hCG in placental trophoblasts.Crosstalk between mitogen-activated protein kinases and mitochondria in cardiac diseases: therapeutic perspectives Baicalein, an active component of Scutellaria baicalensis Georgi, prevents lysophosphatidylcholine-induced cardiac injury by reducing reactive oxygen species production, calcium overload and apoptosis via MAPK pathways.TGF-β2 treatment enhances cytoprotective factors released from embryonic stem cells and inhibits apoptosis in infarcted myocardium.Effects of selective cyclooxygenase-2 and nonselective cyclooxygenase inhibition on myocardial function and perfusion Circadian profiles in the embryonic chick heart: L-type voltage-gated calcium channels and signaling pathways Effects of Wenyangzhenshuai Granule on ERK1/2 and ERK5 activity in the myocardial tissue in a rabbit model of adriamycin-induced chronic heart failure.Phosphatase PTEN is critically involved in post-myocardial infarction remodeling through the Akt/interleukin-10 signaling pathway.Lack of nitric oxide synthases increases lipoprotein immune complex deposition in the aorta and elevates plasma sphingolipid levels in lupus Microparticles from apoptotic RAW 264.7 macrophage cells carry tumour necrosis factor-α functionally active on cardiomyocytes from adult mice.Role of xanthine oxidoreductase in cardiac nitroso-redox imbalance Factors released from embryonic stem cells inhibit apoptosis in H9c2 cells through PI3K/Akt but not ERK pathway.Key pathways associated with heart failure development revealed by gene networks correlated with cardiac remodeling.Effects of probiotic Bacillus as a substitute for antibiotics on antioxidant capacity and intestinal autophagy of piglets.Mitochondria as key targets of cardioprotection in cardiac ischemic disease: role of thyroid hormone triiodothyronine.Cigarette smoke extract induces placental growth factor release from human bronchial epithelial cells via ROS/MAPK (ERK-1/2)/Egr-1 axis.Moxonidine improves cardiac structure and performance in SHR through inhibition of cytokines, p38 MAPK and Akt.Programmed cell death in periodontitis: recent advances and future perspectives.Roles of reactive oxygen species in cell signaling pathways and immune responses to viral infections.Factors released from embryonic stem cells inhibit apoptosis of H9c2 cells.Psoriasis as an Independent Risk Factor for Cardiovascular Disease: An Epidemiologic Analysis Using a National Database.IL-10 counteracts proinflammatory mediator evoked oxidative stress in Caco-2 cells.TNFα shedding in mechanically stressed cardiomyocytes is mediated by Src activation of TACE.Inflammatory mediators in Chinese patients with congestive heart failure.Erythropoietin protects against doxorubicin-induced heart failure.Oleic acid mitigates TNF-α-induced oxidative stress in rat cardiomyocytes.Tyrosol prevents ischemia/reperfusion-induced cardiac injury in H9c2 cells: involvement of ROS, Hsp70, JNK and ERK, and apoptosis.Attenuation of contact hypersensitivity by cell-permeable heat shock protein 70 in BALB/c mouse model.Combined Aerobic and Resistance Exercise Training Improve Hypertension Associated With Menopause

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p38 and ERK1/2 MAPKs mediate the interplay of TNF-alpha and IL-10 in regulating oxidative stress and cardiac myocyte apoptosis.

http://www.wikidata.org/entity/Q46974170

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2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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name

p38 and ERK1/2 MAPKs mediate t ...... and cardiac myocyte apoptosis.

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p38 and ERK1/2 MAPKs mediate t ...... and cardiac myocyte apoptosis.

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type

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p38 and ERK1/2 MAPKs mediate t ...... and cardiac myocyte apoptosis.

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p38 and ERK1/2 MAPKs mediate t ...... and cardiac myocyte apoptosis.

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p38 and ERK1/2 MAPKs mediate t ...... and cardiac myocyte apoptosis.

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AJPHEART.00919.2007

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American Journal of Physiology - Heart and Circulatory Physiology

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p38 and ERK1/2 MAPKs mediate t ...... and cardiac myocyte apoptosis.

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Anita K Sharma

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Dinender K Singla

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Pawan K Singal

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P2860

The apoptosis and proliferation of SAC-activated B cells by IL-10 are associated with changes in Bcl-2, Bcl-xL, and Mcl-1 expression

Signaling by proinflammatory cytokines: oligomerization of TRAF2 and TRAF6 is sufficient for JNK and IKK activation and target gene induction via an amino-terminal effector domain

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

Enhanced TNF alpha and oxidative stress in patients with heart failure: effect of TNF alpha on platelet O2- production

Matrix metalloproteinase-2 contributes to tumor necrosis factor alpha induced apoptosis in cultured rat cardiac myocytes

Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

Adriamycin-induced oxidative stress, activation of MAP kinases and apoptosis in isolated cardiomyocytes.

The inflammatory response in myocardial infarction.

Localization of dichlorofluorescin in cardiac myocytes: implications for assessment of oxidative stress

Cell cycle arrest mediated by the MEK/mitogen-activated protein kinase pathway

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