Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

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A Metadata Analysis of Oxidative Stress Etiology in Preclinical Amyotrophic Lateral Sclerosis: Benefits of Antioxidant Therapy.Shared Functions of Perirhinal and Parahippocampal Cortices: Implications for Cognitive Aging.

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Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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name

Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

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Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

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type

Item

label

Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

@en

Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

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prefLabel

Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

@en

Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

@nl

P1476

Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

@en

P2093

Apoorva Dhanala

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Connor Yee

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Taylor May

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Creative Commons Attribution-NonCommercial 4.0 License

P2860

Modulation of the dynamic instability of tubulin assembly by the microtubule-associated protein tau

Methylene blue reduces aβ levels and rescues early cognitive deficit by increasing proteasome activity

Nicotinamide restores cognition in Alzheimer's disease transgenic mice via a mechanism involving sirtuin inhibition and selective reduction of Thr231-phosphotau

The Role of Amyloid-β Oligomers in Toxicity, Propagation, and Immunotherapy

Genetic reduction of striatal-enriched tyrosine phosphatase (STEP) reverses cognitive and cellular deficits in an Alzheimer's disease mouse model

Physiological and pathological phosphorylation of tau by Cdk5

Neuropathological alterations in Alzheimer disease

The flavonoid quercetin ameliorates Alzheimer's disease pathology and protects cognitive and emotional function in aged triple transgenic Alzheimer's disease model mice

Administration of a selective β2 adrenergic receptor antagonist exacerbates neuropathology and cognitive deficits in a mouse model of Alzheimer's disease

Amitriptyline-mediated cognitive enhancement in aged 3×Tg Alzheimer's disease mice is associated with neurogenesis and neurotrophic activity

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scholarly article

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10.3233/JAD-170490

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Cassie S Mitchell

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2017-11-14T00:00:00Z

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Alzheimer's disease

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membrane proteins

cognitive dysfunction

cytoskeletal proteins

nerve tissue proteins

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5734131

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Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

about

P2860

P2860

Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.

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