Prostaglandin I2 is responsible for ameliorating prostaglandin E2 stress in stimulating the expression of tumor necrosis factor α in a β-amyloid protein -dependent mechanism.

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Prostaglandin I2 is responsible for ameliorating prostaglandin E2 stress in stimulating the expression of tumor necrosis factor α in a β-amyloid protein -dependent mechanism.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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Prostaglandin I2 is responsibl ...... protein -dependent mechanism.

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Prostaglandin I2 is responsibl ...... protein -dependent mechanism.

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Prostaglandin I2 is responsibl ...... protein -dependent mechanism.

@en

Prostaglandin I2 is responsibl ...... protein -dependent mechanism.

@nl

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Prostaglandin I2 is responsibl ...... protein -dependent mechanism.

@en

Prostaglandin I2 is responsibl ...... protein -dependent mechanism.

@nl

P1476

Prostaglandin I2 is responsibl ...... protein -dependent mechanism.

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P2093

Chen-Di Lu

http://www.w3.org/2001/XMLSchema#string

Pu Wang

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Shao-Qin Zheng

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Zi-Yi Gong

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P2860

No advantage of A beta 42-lowering NSAIDs for prevention of Alzheimer dementia in six pooled cohort studies

Role of pro-inflammatory cytokines released from microglia in Alzheimer's disease

Beta-amyloid protein structure determines the nature of cytokine release from rat microglia.

Magnesium ion influx reduces neuroinflammation in Aβ precursor protein/Presenilin 1 transgenic mice by suppressing the expression of interleukin-1β.

Shear-induced interleukin-6 synthesis in chondrocytes: roles of E prostanoid (EP) 2 and EP3 in cAMP/protein kinase A- and PI3-K/Akt-dependent NF-kappaB activation.

Interleukin-6 synthesis in human chondrocytes is regulated via the antagonistic actions of prostaglandin (PG)E2 and 15-deoxy-Δ(12,14)-PGJ2.

Pyrroloquinoline quinone (PQQ) inhibits lipopolysaccharide induced inflammation in part via downregulated NF-κB and p38/JNK activation in microglial and attenuates microglia activation in lipopolysaccharide treatment mice.

Human astrocytes: secretome profiles of cytokines and chemokines.

Response of chondrocytes to shear stress: antagonistic effects of the binding partners Toll-like receptor 4 and caveolin-1.

The induction of cyclooxygenase-2 in IL-1beta-treated endothelial cells is inhibited by prostaglandin E2 through cAMP

P304

102801-102819

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P31

scholarly article

P356

10.18632/ONCOTARGET.18462

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P407

English

P433

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-06-13T00:00:00Z

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P698

29262525

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P932

5732691

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