Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.

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S100A8/A9 Drives Neuroinflammatory Priming and Protects against Anxiety-like Behavior after Sepsis.

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Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.

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http://www.wikidata.org/entity/Q47414857

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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Receptor for advanced glycatio ...... ion, and cognitive impairment.

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Receptor for advanced glycatio ...... ion, and cognitive impairment.

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Receptor for advanced glycatio ...... ion, and cognitive impairment.

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Receptor for advanced glycatio ...... ion, and cognitive impairment.

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Receptor for advanced glycatio ...... ion, and cognitive impairment.

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Receptor for advanced glycatio ...... ion, and cognitive impairment.

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P1476

Receptor for advanced glycatio ...... tion, and cognitive impairment

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P2093

Amanda V Steckert

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Beatriz Sonai

http://www.w3.org/2001/XMLSchema#string

Camila T Ribeiro

http://www.w3.org/2001/XMLSchema#string

Carolina S Girardi

http://www.w3.org/2001/XMLSchema#string

Daniel P Gelain

http://www.w3.org/2001/XMLSchema#string

Felipe Dal-Pizzol

http://www.w3.org/2001/XMLSchema#string

José C F Moreira

http://www.w3.org/2001/XMLSchema#string

João Quevedo

http://www.w3.org/2001/XMLSchema#string

Mariane Rocha

http://www.w3.org/2001/XMLSchema#string

Monique Michels

http://www.w3.org/2001/XMLSchema#string

P2860

RAGE-mediated signaling contributes to intraneuronal transport of amyloid-beta and neuronal dysfunction

Long-term cognitive impairment and functional disability among survivors of severe sepsis

A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye binding

The blood-brain barrier in systemic inflammation

The Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3)

Tau Protein Hyperphosphorylation and Aggregation in Alzheimer's Disease and Other Tauopathies, and Possible Neuroprotective Strategies

The Role of Oxidative Stress-Induced Epigenetic Alterations in Amyloid-β Production in Alzheimer's Disease

Who fans the flames of Alzheimer's disease brains? Misfolded tau on the crossroad of neurodegenerative and inflammatory pathways

Phosphorylation of mammalian target of rapamycin (mTOR) at Ser-2448 is mediated by p70S6 kinase

A multimodal RAGE-specific inhibitor reduces amyloid β-mediated brain disorder in a mouse model of Alzheimer disease

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226-244

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scholarly article

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10.1074/JBC.M117.786756

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English

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293

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Juciano Gasparotto

Tatiana Barichello

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2017-11-10T00:00:00Z

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29127203

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sepsis

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5766916

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Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.

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Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment.

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