Retinal interneuron survival requires non-cell-autonomous Atrx activity.

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Recovery from impaired muscle growth arises from prolonged postnatal accretion of myonuclei in Atrx mutant mice.

P2860

Q47134366-228C1A2E-FF43-4820-AF79-68A954B2F240

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Retinal interneuron survival requires non-cell-autonomous Atrx activity.

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http://www.wikidata.org/entity/Q47806032

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2016 nî lūn-bûn

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2016年学术文章

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Retinal interneuron survival requires non-cell-autonomous Atrx activity.

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Retinal interneuron survival requires non-cell-autonomous Atrx activity.

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Item

label

Retinal interneuron survival requires non-cell-autonomous Atrx activity.

@en

Retinal interneuron survival requires non-cell-autonomous Atrx activity.

@nl

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Retinal interneuron survival requires non-cell-autonomous Atrx activity.

@en

Retinal interneuron survival requires non-cell-autonomous Atrx activity.

@nl

P1476

Retinal interneuron survival requires non-cell-autonomous Atrx activity.

@en

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Adam N Baker

http://www.w3.org/2001/XMLSchema#string

Brandon Y H Zhao

http://www.w3.org/2001/XMLSchema#string

Catherine Tsilfidis

http://www.w3.org/2001/XMLSchema#string

Chantal F Medina

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David J Picketts

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Keqin Yan

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Pamela S Lagali

http://www.w3.org/2001/XMLSchema#string

Stuart G Coupland

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Valerie A Wallace

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P2860

The SWI/SNF protein ATRX co-regulates pseudoautosomal genes that have translocated to autosomes in the mouse genome

Localization of a putative transcriptional regulator (ATRX) at pericentromeric heterochromatin and the short arms of acrocentric chromosomes

Mutations of the X-linked genes encoding neuroligins NLGN3 and NLGN4 are associated with autism

Mutations in a putative global transcriptional regulator cause X-linked mental retardation with alpha-thalassemia (ATR-X syndrome)

ATRX dysfunction induces replication defects in primary mouse cells

X-linked mental retardation and autism are associated with a mutation in the NLGN4 gene, a member of the neuroligin family

Analyzing real-time PCR data by the comparative C(T) method

DAXX/ATRX, MEN1, and mTOR pathway genes are frequently altered in pancreatic neuroendocrine tumors

Altered visual function and interneuron survival in Atrx knockout mice: inference for the human syndrome

Defective survival of proliferating Sertoli cells and androgen receptor function in a mouse model of the ATR-X syndrome

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4787-4803

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scholarly article

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10.1093/HMG/DDW306

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2016-11-01T00:00:00Z

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28173139

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Retinal interneuron survival requires non-cell-autonomous Atrx activity.

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Retinal interneuron survival requires non-cell-autonomous Atrx activity.

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P1433

P1476

P2093

P2860

P304

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P356

P433

P478

P577

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P698