about
The transient intraluminal filament middle cerebral artery occlusion model as a model of endovascular thrombectomy in strokeNeuroprotection for stroke: current status and future perspectivesThe exact science of stroke thrombolysis and the quiet art of patient selectionCapillary pericytes regulate cerebral blood flow in health and disease.The contribution of L-arginine to the neurotoxicity of recombinant tissue plasminogen activator following cerebral ischemia: a review of rtPA neurotoxicityLaser Doppler flowmetry to measure changes in cerebral blood flow.Importance of preclinical research in the development of neuroprotective strategies for ischemic stroke.The role of antioxidants in models of inflammation: emphasis on L-arginine and arachidonic acid metabolism.Mechanisms of action of green tea catechins, with a focus on ischemia-induced neurodegeneration.Alteplase treatment does not increase brain injury after mechanical middle cerebral artery occlusion in the rat.A systematic review and meta-analysis of randomized controlled trials of endovascular thrombectomy compared with best medical treatment for acute ischemic stroke.A Critical Role for Astrocytes in Hypercapnic Vasodilation in Brain.Cerebral blood flow alteration in neuroprotection following cerebral ischaemia.Neuroprotection for ischaemic stroke: translation from the bench to the bedside.Thrombolytic agents for acute ischaemic stroke treatment: the past, present and future.Complications associated with recombinant tissue plasminogen activator therapy for acute ischaemic stroke.The IMPROVE Guidelines (Ischaemia Models: Procedural Refinements Of in Vivo Experiments).Multi-modal assessment of neurovascular coupling during cerebral ischaemia and reperfusion using remote middle cerebral artery occlusion.Melatonin treatment following stroke induction modulates L-arginine metabolism.Novel method to study pericyte contractility and responses to ischaemia in vitro using electrical impedance.Differential effects of paracrine factors on the survival of cells of the neurovascular unit during oxygen glucose deprivation.The role of the endoplasmic reticulum stress response following cerebral ischemia.The life of Bo K. Siesjö, MD, PhD, 1930-2013.Cerebral hypoxia-ischemia and middle cerebral artery occlusion induce expression of the cannabinoid CB2 receptor in the brain.The future of stroke therapy must not be mired by past arguments.Cerebral heme oxygenase 1 and 2 spatial distribution is modulated following injury from hypoxia-ischemia and middle cerebral artery occlusion in rats.Reply: Intravenous thrombolysis for ischaemic strokes: a call for reappraisal.Rapamycin in ischemic stroke: Old drug, new tricks?Tin protoporphyrin provides protection following cerebral hypoxia-ischemia: Involvement of alternative pathwaysInvestigation of the novel mTOR inhibitor AZD2014 in neuronal ischemia.Characterization of a rat hypoxia-ischemia model where duration of hypoxia is determined by seizure activityReply: Thrombolysis in acute ischaemic strokeTranscranial contrast-enhanced ultrasound in the rat brain reveals substantial hyperperfusion acutely post-strokeMetabolic-vascular coupling in skeletal muscle: A potential role for capillary pericytes?Harnessing the stem cell properties of pericytes to repair the brainPericytes and Neurovascular Function in the Healthy and Diseased BrainApplications of Nanotechnology in the Diagnosis and Therapy of StrokeThe effect of rapamycin treatment on cerebral ischemia: A systematic review and meta-analysis of animal model studies
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Brad A Sutherland
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Brad A Sutherland
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Brad A. Sutherland
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Brad A. Sutherland
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Brad A Sutherland
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Brad A Sutherland
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Brad A. Sutherland
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Brad A. Sutherland
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Brad A Sutherland
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Brad A Sutherland
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Brad A. Sutherland
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Brad A. Sutherland
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