Changes in biochemical processes in cerebellar granule cells of mice exposed to methylmercury.

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Comparison of alterations in amino acids content in cultured astrocytes or neurons exposed to methylmercury separately or in co-culture The effect of methylmercury exposure on behavior and cerebellar granule cell physiology in aged mice.

P2860

Q37188541-301615AC-1791-4045-B472-623A1B25C004 Q42511107-8B4E3B89-647F-40EF-A31E-281C4738A2F6

P2860

Changes in biochemical processes in cerebellar granule cells of mice exposed to methylmercury.

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http://www.wikidata.org/entity/Q48138134

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2007 nî lūn-bûn

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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2007年學術文章

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Changes in biochemical process ...... mice exposed to methylmercury.

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Changes in biochemical process ...... mice exposed to methylmercury.

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Changes in biochemical process ...... mice exposed to methylmercury.

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Changes in biochemical process ...... mice exposed to methylmercury.

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Changes in biochemical process ...... mice exposed to methylmercury.

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Changes in biochemical process ...... mice exposed to methylmercury.

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P1476

Changes in biochemical process ...... mice exposed to methylmercury.

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Bhupinder Bawa

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Kerry A Thuett

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Louise C Abbott

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Sairam Bellum

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P2860

Methylmercury poisoning in Iraq

Translocation of apoptosis-inducing factor in cerebellar granule cells exposed to neurotoxic agents inducing oxidative stress

The biochemistry of apoptosis

Nuclear calcium: a key regulator of gene expression.

Apoptosis in neurodegenerative disorders.

Fluoro-Jade: a novel fluorochrome for the sensitive and reliable histochemical localization of neuronal degeneration.

Mercuric compounds inhibit human monocyte function by inducing apoptosis: evidence for formation of reactive oxygen species, development of mitochondrial membrane permeability transition and loss of reductive reserve.

Methylmercury cytotoxicity in PC12 cells is mediated by primary glutathione depletion independent of excess reactive oxygen species generation.

Assessment of mercury concentrations in mouse brain using different routes of administration and different tissue preparations.

Acute exposure to methylmercury opens the mitochondrial permeability transition pore in rat cerebellar granule cells.

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261-269

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scholarly article

P356

10.1080/10915810701369758

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P478

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George Stoica

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2007-05-01T00:00:00Z

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6271973

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17564908

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Changes in biochemical processes in cerebellar granule cells of mice exposed to methylmercury.

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P2860

P2860

Changes in biochemical processes in cerebellar granule cells of mice exposed to methylmercury.

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type

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P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P50

P577

P5875

P698