about
Arterially perfused neurosphere-derived cells distribute outside the ischemic core in a model of transient focal ischemia and reperfusion in vitroCaspase-3 contributes to ZO-1 and Cl-5 tight-junction disruption in rapid anoxic neurovascular unit damage.Molecular anatomy of the cerebral microvessels in the isolated guinea-pig brain.Different permeability of potassium salts across the blood-brain barrier follows the Hofmeister seriesModerate hypoxia followed by reoxygenation results in blood-brain barrier breakdown via oxidative stress-dependent tight-junction protein disruption.The in vitro isolated whole guinea pig brain as a model to study epileptiform activity patterns.Kainic acid-induced albumin leak across the blood-brain barrier facilitates epileptiform hyperexcitability in limbic regions.Discharge threshold is enhanced for several seconds after a single interictal spike in a model of focal epileptogenesis.Epileptiform ictal discharges are prevented by periodic interictal spiking in the olfactory cortex.Penumbra region excitability is not enhanced acutely after cerebral ischemia in the in vitro isolated guinea pig brain.Interneuronal Network Activity at the Onset of Seizure-Like Events in Entorhinal Cortex Slices.Rapid in vitro elimination of anesthetic doses of thiopental in the isolated guinea pig brain.Propagation dynamics of epileptiform activity acutely induced by bicuculline in the hippocampal-parahippocampal region of the isolated Guinea pig brain.Pravastatin acute neuroprotective effects depend on blood brain barrier integrity in experimental cerebral ischemia.Activation of cerebral endothelium is required for mononuclear cell recruitment in a novel in vitro model of brain inflammation.Acute lipophilicity-dependent effect of intravascular simvastatin in the early phase of focal cerebral ischemia.Expression of adhesion factors induced by epileptiform activity in the endothelium of the isolated guinea pig brain in vitro.Functional and structural correlates of magnetic resonance patterns in a new in vitro model of cerebral ischemia by transient occlusion of the medial cerebral artery.Seizure-induced brain-borne inflammation sustains seizure recurrence and blood-brain barrier damage.Mechanisms of C-reactive protein-induced blood-brain barrier disruption.Nitric oxide synthase inhibitors unmask acetylcholine-mediated constriction of cerebral vessels in the in vitro isolated guinea-pig brain.
P50
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P50
description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Laura Librizzi
@ast
Laura Librizzi
@en
Laura Librizzi
@es
Laura Librizzi
@nl
Laura Librizzi
@sl
type
label
Laura Librizzi
@ast
Laura Librizzi
@en
Laura Librizzi
@es
Laura Librizzi
@nl
Laura Librizzi
@sl
prefLabel
Laura Librizzi
@ast
Laura Librizzi
@en
Laura Librizzi
@es
Laura Librizzi
@nl
Laura Librizzi
@sl
P1053
J-7889-2016
P106
P1153
6508374418
P21
P31
P3829
P496
0000-0002-2741-4452