Mitochondrial Cyclophilin D as a Potential Therapeutic Target for Ischemia-Induced Facial Palsy in Rats.

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Reduction in Autophagy by (-)-Epigallocatechin-3-Gallate (EGCG): a Potential Mechanism of Prevention of Mitochondrial Dysfunction After Subarachnoid Hemorrhage.

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Mitochondrial Cyclophilin D as a Potential Therapeutic Target for Ischemia-Induced Facial Palsy in Rats.

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Mitochondrial Cyclophilin D as ...... -Induced Facial Palsy in Rats.

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Mitochondrial Cyclophilin D as ...... -Induced Facial Palsy in Rats.

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Mitochondrial Cyclophilin D as ...... -Induced Facial Palsy in Rats.

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Mitochondrial Cyclophilin D as ...... -Induced Facial Palsy in Rats.

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Mitochondrial Cyclophilin D as ...... -Induced Facial Palsy in Rats.

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Mitochondrial Cyclophilin D as ...... -Induced Facial Palsy in Rats.

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P1476

Mitochondrial Cyclophilin D as ...... -Induced Facial Palsy in Rats.

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P2093

Aimin Li

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Chnagtao Liu

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Duanlei Wang

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Huizhen Chen

http://www.w3.org/2001/XMLSchema#string

Jiaqiu Zhu

http://www.w3.org/2001/XMLSchema#string

Jie Yin

http://www.w3.org/2001/XMLSchema#string

Jinwang Xu

http://www.w3.org/2001/XMLSchema#string

Yong Sun

http://www.w3.org/2001/XMLSchema#string

Zhen Chen

http://www.w3.org/2001/XMLSchema#string

Ziyuan Zhang

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P2860

p53 opens the mitochondrial permeability transition pore to trigger necrosis

Complex contribution of cyclophilin D to Ca2+-induced permeability transition in brain mitochondria, with relation to the bioenergetic state

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemia

The targeting of cyclophilin D by RNAi as a novel cardioprotective therapy: evidence from two-photon imaging.

Mitochondrial cyclophilin-D as a critical mediator of ischaemic preconditioning.

Cysteine 203 of cyclophilin D is critical for cyclophilin D activation of the mitochondrial permeability transition pore

CypD(-/-) hearts have altered levels of proteins involved in Krebs cycle, branch chain amino acid degradation and pyruvate metabolism

Caspase inhibitors promote alternative cell death pathways.

Two independent pathways of regulated necrosis mediate ischemia-reperfusion injury

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Mitochondrial Cyclophilin D as a Potential Therapeutic Target for Ischemia-Induced Facial Palsy in Rats.

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Mitochondrial Cyclophilin D as a Potential Therapeutic Target for Ischemia-Induced Facial Palsy in Rats.

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