about
Inhalation of rod-like carbon nanotubes causes unconventional allergic airway inflammationMiR-155 is overexpressed in patients with atopic dermatitis and modulates T-cell proliferative responses by targeting cytotoxic T lymphocyte-associated antigen 4.Attenuated expression of tenascin-C in ovalbumin-challenged STAT4-/- miceTopically applied ZnO nanoparticles suppress allergen induced skin inflammation but induce vigorous IgE production in the atopic dermatitis mouse model.Loss of beta2-integrin-mediated cytoskeletal linkage reprogrammes dendritic cells to a mature migratory phenotype.Acinetobacter species in the skin microbiota protect against allergic sensitization and inflammation.Mice Lacking beta2-Integrin Function Remain Glucose Tolerant in Spite of Insulin Resistance, Neutrophil Infiltration and Inflammation.Anti-Inflammatory Effects of Metformin Irrespective of Diabetes StatusA sensory neuron-expressed IL-31 receptor mediates T helper cell-dependent itch: Involvement of TRPV1 and TRPA1.Optimal T Cell Activation and B Cell Antibody Responses In Vivo Require the Interaction between Leukocyte Function-Associated Antigen-1 and Kindlin-3.The β2 integrin-kindlin-3 interaction is essential for T-cell homing but dispensable for T-cell activation in vivo.Absence of CCR4 exacerbates skin inflammation in an oxazolone-induced contact hypersensitivity model.ST2 regulates allergic airway inflammation and T-cell polarization in epicutaneously sensitized mice.The chemokine receptor CCR3 participates in tissue remodeling during atopic skin inflammation.Filamin A Regulates Neutrophil Adhesion, Production of Reactive Oxygen Species, and Neutrophil Extracellular Trap Release.Toll-like receptor activation during cutaneous allergen sensitization blocks development of asthma through IFN-gamma-dependent mechanisms.Foxp3+ cells control Th2 responses in a murine model of atopic dermatitis.Filamin A Is Required for Optimal T Cell Integrin-Mediated Force Transmission, Flow Adhesion, and T Cell Trafficking.Functional Beta2-Integrins Restrict Skin Inflammation In VivoContact sensitizer 2,4-dinitrochlorobenzene is a highly potent human TRPA1 agonistThe Temporal and Spatial Dynamics of Foxp3+ Treg Cell–Mediated Suppression during Contact Hypersensitivity Responses in a Murine ModelIL-33 and ST2 in Atopic Dermatitis: Expression Profiles and Modulation by Triggering FactorsIntradermal Cytosine-Phosphate-Guanosine Treatment Reduces Lung Inflammation but Induces IFN-γ–Mediated Airway Hyperreactivity in a Murine Model of Natural Rubber Latex AllergyRepeated epicutaneous exposures to ovalbumin progressively induce atopic dermatitis-like skin lesions in miceA β2-Integrin/MRTF-A/SRF Pathway Regulates Dendritic Cell Gene Expression, Adhesion, and Traction Force Generation.Serum chitinase-like protein YKL-40 is linked to small airway function in children with asthmatic symptoms
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Terhi Savinko
@ast
Terhi Savinko
@en
Terhi Savinko
@es
Terhi Savinko
@nl
Terhi Savinko
@sl
type
label
Terhi Savinko
@ast
Terhi Savinko
@en
Terhi Savinko
@es
Terhi Savinko
@nl
Terhi Savinko
@sl
prefLabel
Terhi Savinko
@ast
Terhi Savinko
@en
Terhi Savinko
@es
Terhi Savinko
@nl
Terhi Savinko
@sl
P106
P21
P31
P496
0000-0003-4483-7235
P569
2000-01-01T00:00:00Z