Nitric oxide scavenging by hemoglobin or nitric oxide synthase inhibition by N-nitro-L-arginine induces cortical spreading ischemia when K+ is increased in the subarachnoid space.
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Towards use of MRI-guided ultrasound for treating cerebral vasospasm.The importance of early brain injury after subarachnoid hemorrhagePathophysiology of the neurovascular unit: disease cause or consequence?Spreading Depression, Spreading Depolarizations, and the Cerebral VasculatureThe role of spreading depolarization in subarachnoid hemorrhageIschaemia triggered by spreading neuronal activation is inhibited by vasodilators in ratsSpreading depolarizations cycle around and enlarge focal ischaemic brain lesionsDistinct spatiotemporal patterns of spreading depolarizations during early infarct evolution: evidence from real-time imaging.Dynamic in vivo imaging of cerebral blood flow and blood-brain barrier permeability.Impaired neurovascular coupling to ictal epileptic activity and spreading depolarization in a patient with subarachnoid hemorrhage: possible link to blood-brain barrier dysfunction.Detecting tissue deterioration after brain injury: regional blood flow level versus capacity to raise blood flowNormobaric hyperoxia improves cerebral blood flow and oxygenation, and inhibits peri-infarct depolarizations in experimental focal ischaemia.Spreading depression in the brainstem of the adult rat: electrophysiological parameters and influences on regional brainstem blood flow.Cortical spreading ischaemia is a novel process involved in ischaemic damage in patients with aneurysmal subarachnoid haemorrhageBiphasic direct current shift, haemoglobin desaturation and neurovascular uncoupling in cortical spreading depression.Cyclosporine A, FK506, and NIM811 ameliorate prolonged CBF reduction and impaired neurovascular coupling after cortical spreading depression.Recurrent spreading depolarizations after subarachnoid hemorrhage decreases oxygen availability in human cerebral cortex.Recurrent spontaneous spreading depolarizations facilitate acute dendritic injury in the ischemic penumbraHigher brain extracellular potassium is associated with brain metabolic distress and poor outcome after aneurysmal subarachnoid hemorrhage.Uncoupling of endothelial nitric oxide synthase after experimental subarachnoid hemorrhage.Imaging reveals the focal area of spreading depolarizations and a variety of hemodynamic responses in a rat microembolic stroke model.Delayed cerebral ischemia and spreading depolarization in absence of angiographic vasospasm after subarachnoid hemorrhage.A role of the sodium pump in spreading ischemia in rats.Advancing age and ischemia elevate the electric threshold to elicit spreading depolarization in the cerebral cortex of young adult rats.Neuroprotection in subarachnoid hemorrhageRequisite ischemia for spreading depolarization occurrence after subarachnoid hemorrhage in rodents.Large field-of-view movement-compensated intrinsic optical signal imaging for the characterization of the haemodynamic response to spreading depolarizations in large gyrencephalic brains.Multifaceted roles for astrocytes in spreading depolarization: A target for limiting spreading depolarization in acute brain injury?Is spreading depolarization characterized by an abrupt, massive release of gibbs free energy from the human brain cortex?Clinical trials in cardiac arrest and subarachnoid hemorrhage: lessons from the past and ideas for the future.Subarachnoid hemorrhage, spreading depolarizations and impaired neurovascular coupling.Peri-infarct depolarizations during focal ischemia in the awake Spontaneously Hypertensive Rat. Minimizing anesthesia confounds in experimental strokeGenetic elimination of eNOS reduces secondary complications of experimental subarachnoid hemorrhage.Microthrombosis after aneurysmal subarachnoid hemorrhage: an additional explanation for delayed cerebral ischemia.Cerebral vasospasm following subarachnoid hemorrhage: time for a new world of thought.Controversies and evolving new mechanisms in subarachnoid hemorrhage.Genetic determinants of cerebral vasospasm, delayed cerebral ischemia, and outcome after aneurysmal subarachnoid hemorrhage.Spreading depolarization: a possible new culprit in the delayed cerebral ischemia of subarachnoid hemorrhage.Clinical relevance of cortical spreading depression in neurological disorders: migraine, malignant stroke, subarachnoid and intracranial hemorrhage, and traumatic brain injury.Effect of pharmaceutical treatment on vasospasm, delayed cerebral ischemia, and clinical outcome in patients with aneurysmal subarachnoid hemorrhage: a systematic review and meta-analysis.
P2860
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P2860
Nitric oxide scavenging by hemoglobin or nitric oxide synthase inhibition by N-nitro-L-arginine induces cortical spreading ischemia when K+ is increased in the subarachnoid space.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Nitric oxide scavenging by hem ...... sed in the subarachnoid space.
@en
Nitric oxide scavenging by hem ...... sed in the subarachnoid space.
@nl
type
label
Nitric oxide scavenging by hem ...... sed in the subarachnoid space.
@en
Nitric oxide scavenging by hem ...... sed in the subarachnoid space.
@nl
prefLabel
Nitric oxide scavenging by hem ...... sed in the subarachnoid space.
@en
Nitric oxide scavenging by hem ...... sed in the subarachnoid space.
@nl
P2093
P2860
P1476
Nitric oxide scavenging by hem ...... sed in the subarachnoid space.
@en
P2093
Einhäupl KM
Lauritzen M
Lindauer U
P2860
P304
P356
10.1097/00004647-199809000-00007
P577
1998-09-01T00:00:00Z
P5875
P6179
1034061690