Loss of Maged1 results in obesity, deficits of social interactions, impaired sexual behavior and severe alteration of mature oxytocin production in the hypothalamus.
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Oxytocin and cardioprotection in diabetes and obesityFrom Autism to Eating Disorders and More: The Role of Oxytocin in Neuropsychiatric DisordersObesity in children with autism spectrum disorderThe neurobiology of the Prader-Willi phenotype of fragile X syndromeSystems genetic analysis of osteoblast-lineage cellsXp11.22 deletions encompassing CENPVL1, CENPVL2, MAGED1 and GSPT2 as a cause of syndromic X-linked intellectual disability.Tryptophan and kynurenic Acid may produce an amplified effect in central fatigue induced by chronic sleep disorder.Intergenerational genomic DNA methylation patterns in mouse hybrid strains.Role of oxytocin signaling in the regulation of body weight.Oxytocin and vasopressin systems in genetic syndromes and neurodevelopmental disordersOntogenesis of oxytocin pathways in the mammalian brain: late maturation and psychosocial disordersA mouse model of L-2-hydroxyglutaric aciduria, a disorder of metabolite repair.Oxytocin differentially affects sucrose taking and seeking in male and female ratsMedial nucleus tractus solitarius oxytocin receptor signaling and food intake control: the role of gastrointestinal satiation signal processing.Oxytocin, feeding, and satiety.Oxytocin and Animal Models for Autism Spectrum Disorder.Complex roles of NRAGE on tumor.A Comprehensive Guide to the MAGE Family of Ubiquitin Ligases.Maged1 co-interacting with CREB through a hexapeptide repeat domain regulates learning and memory in mice.Oxytocin and Prader-Willi Syndrome.MAGED1 is a novel regulator of a select subset of bHLH PAS transcription factors.Muscle dysfunction caused by loss of Magel2 in a mouse model of Prader-Willi and Schaaf-Yang syndromes.Oxytocin treatment in children with Prader-Willi syndrome: A double-blind, placebo-controlled, crossover study.Whole-genome resequencing reveals candidate mutations for pig prolificacy.Interplay Between the Gut-Brain Axis, Obesity and Cognitive Function.Oxytocin as a Modulator of Synaptic Plasticity: Implications for Neurodevelopmental Disorders.Deletion of in mice abolishes locomotor and reinforcing effects of cocaine
P2860
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P2860
Loss of Maged1 results in obesity, deficits of social interactions, impaired sexual behavior and severe alteration of mature oxytocin production in the hypothalamus.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
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2012年學術文章
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name
Loss of Maged1 results in obes ...... roduction in the hypothalamus.
@en
Loss of Maged1 results in obes ...... roduction in the hypothalamus.
@nl
type
label
Loss of Maged1 results in obes ...... roduction in the hypothalamus.
@en
Loss of Maged1 results in obes ...... roduction in the hypothalamus.
@nl
prefLabel
Loss of Maged1 results in obes ...... roduction in the hypothalamus.
@en
Loss of Maged1 results in obes ...... roduction in the hypothalamus.
@nl
P2093
P2860
P356
P1476
Loss of Maged1 results in obes ...... roduction in the hypothalamus.
@en
P2093
Carlos Dombret
Hélène Hardin-Pouzet
Jacques L Michaud
Mathieu J M Bertrand
Olivier De Backer
Olivier Schakman
Tuan Nguyen
P2860
P304
P356
10.1093/HMG/DDS310
P577
2012-08-02T00:00:00Z