The evolving field of salt transport regulation in the Steve Hebert Lecture.

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The evolving field of salt transport regulation in the Steve Hebert Lecture.

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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2016年學術文章

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2016年學術文章

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The evolving field of salt transport regulation in the Steve Hebert Lecture.

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The evolving field of salt transport regulation in the Steve Hebert Lecture.

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The evolving field of salt transport regulation in the Steve Hebert Lecture.

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The evolving field of salt transport regulation in the Steve Hebert Lecture.

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The evolving field of salt transport regulation in the Steve Hebert Lecture.

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The evolving field of salt transport regulation in the Steve Hebert Lecture.

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P1476

The evolving field of salt transport regulation in the Steve Hebert Lecture.

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P2860

KLHL3 mutations cause familial hyperkalemic hypertension by impairing ion transport in the distal nephron

Gitelman's variant of Bartter's syndrome, inherited hypokalaemic alkalosis, is caused by mutations in the thiazide-sensitive Na-Cl cotransporter

The WNK-regulated SPAK/OSR1 kinases directly phosphorylate and inhibit the K+-Cl- co-transporters

Kelch-like 3 and Cullin 3 regulate electrolyte homeostasis via ubiquitination and degradation of WNK4

Chloride Sensing by WNK1 Involves Inhibition of Autophosphorylation

Human hypertension caused by mutations in WNK kinases

Pathophysiology of functional mutations of the thiazide-sensitive Na-Cl cotransporter in Gitelman disease

The Na+:Cl- cotransporter is activated and phosphorylated at the amino-terminal domain upon intracellular chloride depletion

Activation of the thiazide-sensitive Na+-Cl- cotransporter by the WNK-regulated kinases SPAK and OSR1

Cloning and expression of an inwardly rectifying ATP-regulated potassium channel

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F68-70

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scholarly article

editorial

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10.1152/AJPRENAL.00229.2016

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311

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Gerardo Gamba

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2016-05-04T00:00:00Z

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27147671

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