TNF-α Increases Production of Reactive Oxygen Species through Cdk5 Activation in Nociceptive Neurons.

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TNF-α Increases Production of Reactive Oxygen Species through Cdk5 Activation in Nociceptive Neurons.

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2018 nî lūn-bûn

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TNF-α Increases Production of ...... vation in Nociceptive Neurons.

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TNF-α Increases Production of ...... vation in Nociceptive Neurons.

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TNF-α Increases Production of ...... vation in Nociceptive Neurons.

@en

TNF-α Increases Production of ...... vation in Nociceptive Neurons.

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TNF-α Increases Production of ...... vation in Nociceptive Neurons.

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TNF-α Increases Production of ...... vation in Nociceptive Neurons.

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P1476

TNF-α Increases Production of ...... ivation in Nociceptive Neurons

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P2093

Christian González-Billault

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Franco Ferrari

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Pablo Lazcano

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Rodrigo Sandoval

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P2860

Cyclin-dependent kinase 5 modulates nociceptive signaling through direct phosphorylation of transient receptor potential vanilloid 1

Molecular targeting of NOX4 for neuropathic pain after traumatic injury of the spinal cord.

Reactive oxygen species in inflammation and tissue injury

Roscovitine targets, protein kinases and pyridoxal kinase

Cellular and Molecular Mechanisms of Pain

Molecular insights of p47phox phosphorylation dynamics in the regulation of NADPH oxidase activation and superoxide production.

Advanced oxidation protein products sensitized the transient receptor potential vanilloid 1 via NADPH oxidase 1 and 4 to cause mechanical hyperalgesia.

Advanced Oxidative Protein Products Cause Pain Hypersensitivity in Rats by Inducing Dorsal Root Ganglion Neurons Apoptosis via NADPH Oxidase 4/c-Jun N-terminal Kinase Pathways.

Cyclin-dependent kinase 5 activity regulates pain signaling.

Molecular Roles of Cdk5 in Pain Signaling

P304

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scholarly article

P356

10.3389/FPHYS.2018.00065

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P478

http://www.w3.org/2001/XMLSchema#string

P50

Elías Utreras

Nicolás Pinto-Pardo

P577

2018-02-06T00:00:00Z

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P698

29467671

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P932

5808211

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