Sustained CaMKII activity mediates transient oxidative stress-induced long-term facilitation of L-type Ca(2+) current in cardiomyocytes.
about
CaMKII oxidative activation and the pathogenesis of cardiac diseaseMitochondrial ion channels/transporters as sensors and regulators of cellular redox signaling.Hydrogen peroxide sensing and signaling by protein kinases in the cardiovascular system.Resveratrol protects rabbit ventricular myocytes against oxidative stress-induced arrhythmogenic activity and Ca2+ overload.A Memory Molecule, Ca(2+)/Calmodulin-Dependent Protein Kinase II and Redox Stress; Key Factors for Arrhythmias in a Diseased Heart.Mitochondrial Ca2+ and regulation of the permeability transition pore.
P2860
Sustained CaMKII activity mediates transient oxidative stress-induced long-term facilitation of L-type Ca(2+) current in cardiomyocytes.
description
2011 nî lūn-bûn
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2011年の論文
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年学术文章
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2011年學術文章
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name
Sustained CaMKII activity medi ...... term facilitation of L-type Ca
@nl
Sustained CaMKII activity medi ...... 2+) current in cardiomyocytes.
@en
type
label
Sustained CaMKII activity medi ...... term facilitation of L-type Ca
@nl
Sustained CaMKII activity medi ...... 2+) current in cardiomyocytes.
@en
prefLabel
Sustained CaMKII activity medi ...... term facilitation of L-type Ca
@nl
Sustained CaMKII activity medi ...... 2+) current in cardiomyocytes.
@en
P2093
P1476
Sustained CaMKII activity medi ...... (2+) current in cardiomyocytes
@en
P2093
Shin-Young Ryu
Sun-Hyun Park
Won-Kyung Ho
Young-Hwan Song
P304
P356
10.1016/J.FREERADBIOMED.2011.07.022
P50
P577
2011-08-05T00:00:00Z