Progesterone/RANKL is a major regulatory axis in the human breast. - Test2 - elhamkhani - TriplyDB

Progesterone/RANKL is a major regulatory axis in the human breast.

Progesterone/RANKL is a major regulatory axis in the human breast.

http://www.wikidata.org/entity/Q51042996

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Progestin effects on cell proliferation pathways in the postmenopausal mammary gland Steroid hormones, steroid receptors, and breast cancer stem cells Progesterone and Overlooked Endocrine Pathways in Breast Cancer Pathogenesis Form and function: how estrogen and progesterone regulate the mammary epithelial hierarchy The mammary stem cell hierarchy: a looking glass into heterogeneous breast cancer landscapes Where Do Bone-Targeted Agents RANK in Breast Cancer Treatment?Targeting RANKL in metastasis The Roles of Epithelial-to-Mesenchymal Transition (EMT) and Mesenchymal-to-Epithelial Transition (MET) in Breast Cancer Bone Metastasis: Potential Targets for Prevention and Treatment Molecular determinants of context-dependent progesterone receptor action in breast cancer Growth of human breast tissues from patient cells in 3D hydrogel scaffolds ELF5 Drives Lung Metastasis in Luminal Breast Cancer through Recruitment of Gr1+ CD11b+ Myeloid-Derived Suppressor Cells Deciphering the divergent roles of progestogens in breast cancer.Posttranslationally modified progesterone receptors direct ligand-specific expression of breast cancer stem cell-associated gene programs Minireview: Progesterone Regulation of Proliferation in the Normal Human Breast and in Breast Cancer: A Tale of Two Scenarios?BRCA1 haploinsufficiency cell-autonomously activates RANKL expression and generates denosumab-responsive breast cancer-initiating cells.Aldehyde dehydrogenase and estrogen receptor define a hierarchy of cellular differentiation in the normal human mammary epithelium.Cyclin A2 and its associated kinase activity are required for optimal induction of progesterone receptor target genes in breast cancer cells Biology of the RANKL-RANK-OPG System in Immunity, Bone, and Beyond.Tracking progesterone receptor-mediated actions in breast cancer Human breast progenitor cell numbers are regulated by WNT and TBX3.Dissecting the Biology of Menstrual Cycle-Associated Breast Cancer Risk.A role for site-specific phosphorylation of mouse progesterone receptor at serine 191 in vivo.Progesterone action in breast, uterine, and ovarian cancers.An ex vivo model to study hormone action in the human breast.Progesterone and Wnt4 control mammary stem cells via myoepithelial crosstalk Progesterone downregulation of miR-141 contributes to expansion of stem-like breast cancer cells through maintenance of progesterone receptor and Stat5a.Cervical (pre)neoplastic microenvironment promotes the emergence of tolerogenic dendritic cells via RANKL secretion.Effects of Combination of Estradiol with Selective Progesterone Receptor Modulators (SPRMs) on Human Breast Cancer Cells In Vitro and In Vivo.Quantification of regenerative potential in primary human mammary epithelial cells.RANK Signaling Amplifies WNT-Responsive Mammary Progenitors through R-SPONDIN1.Cripto-1 ablation disrupts alveolar development in the mouse mammary gland through a progesterone receptor-mediated pathway.Osteoprotegerin and breast cancer risk by hormone receptor subtype: a nested case-control study in the EPIC cohort.Propagation of oestrogen receptor-positive and oestrogen-responsive normal human breast cells in culture.A Common Docking Domain in Progesterone Receptor-B links DUSP6 and CK2 signaling to proliferative transcriptional programs in breast cancer cells Genomic Biomarkers for Breast Cancer Risk.Progesterone receptor and Stat5 signaling cross talk through RANKL in mammary epithelial cells.Prevention of breast cancer by RANKL/RANK blockade.BRCA and Early Events in the Development of Serous Ovarian Cancer.The prognostic role of RANK SNP rs34945627 in breast cancer patients with bone metastases.A DNA segment spanning the mouse Tnfsf11 transcription unit and its upstream regulatory domain rescues the pleiotropic biologic phenotype of the RANKL null mouse.

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Progesterone/RANKL is a major regulatory axis in the human breast.

http://www.wikidata.org/entity/Q51042996

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年學術文章

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name

Progesterone/RANKL is a major regulatory axis in the human breast.

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Progesterone/RANKL is a major regulatory axis in the human breast.

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type

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Progesterone/RANKL is a major regulatory axis in the human breast.

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Progesterone/RANKL is a major regulatory axis in the human breast.

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Progesterone/RANKL is a major regulatory axis in the human breast.

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Progesterone/RANKL is a major regulatory axis in the human breast.

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Science Translational Medicine

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Progesterone/RANKL is a major regulatory axis in the human breast.

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Ayyakkannu Ayyanan

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Cathrin Brisken

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Jean-Francois Delaloye

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Maria Gutierrez

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Maryse Fiche

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Tamara Tanos

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Wassim Raffoul

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William Dougall

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P2860

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

Hormone action in the mammary gland

Osteoclast differentiation factor RANKL controls development of progestin-driven mammary cancer

Estrogen receptor alpha mediates progestin-induced mammary tumor growth by interacting with progesterone receptors at the cyclin D1/MYC promoters

The osteoclast differentiation factor osteoprotegerin-ligand is essential for mammary gland development

RANK ligand mediates progestin-induced mammary epithelial proliferation and carcinogenesis

Evaluation of cell cycle arrest in estrogen responsive MCF-7 breast cancer cells: pitfalls of the MTS assay

Amphiregulin is an essential mediator of estrogen receptor alpha function in mammary gland development

Defective mammary gland morphogenesis in mice lacking the progesterone receptor B isoform

Cytoscape 2.8: new features for data integration and network visualization

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182

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Pascal Schneider

Pablo C Echeverria

Ozden Yalcin-Ozuysal

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