about
Chronic arthritis leads to disturbances in the bone collagen network.Upregulation of inflammatory genes and downregulation of sclerostin gene expression are key elements in the early phase of fragility fracture healing.Identification of a cytokine network sustaining neutrophil and Th17 activation in untreated early rheumatoid arthritis.Soluble receptor activator of nuclear factor κB ligand/osteoprotegerin ratio is increased in systemic lupus erythematosus patients.Effect of Tumor Necrosis Factor Inhibitor Therapy on Osteoclasts Precursors in Ankylosing SpondylitisPachydermoperiostosis in an African patient caused by a Chinese/Japanese SLCO2A1 mutation-case report and review of literature.Methotrexate and low-dose prednisolone downregulate osteoclast function by decreasing receptor activator of nuclear factor-κβ expression in monocytes from patients with early rheumatoid arthritis.Effect of Tumor Necrosis Factor Inhibitor Therapy on Osteoclasts Precursors in Rheumatoid Arthritis.Low osteocalcin/collagen type I bone gene expression ratio is associated with hip fragility fractures.Lymphotoxin-α 252 A>G polymorphism: a link between disease susceptibility and dyslipidemia in rheumatoid arthritis?Cytokine pattern in very early rheumatoid arthritis favours B-cell activation and survival.Rheumatoid arthritis bone fragility is associated with upregulation of IL17 and DKK1 gene expression.TNF promoter -308 G>A and LTA 252 A>G polymorphisms in Portuguese patients with systemic lupus erythematosusThe in vitro behaviour of canine osteoblasts derived from different bone typesIsolation and Generation of Osteoblasts
P50
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P50
description
hulumtuese
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onderzoeker
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researcher
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հետազոտող
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Perpetuo I
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Perpetuo IP
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Perpétuo I
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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Inês P Perpétuo
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P1053
F-3643-2011
P106
P21
P2798
P31
P3829
P496
0000-0001-7671-2539