Further evidence for BRCA1 communication with the inactive X chromosome.
about
BRCA1 protein and nucleolin colocalize in breast carcinoma tissue and cancer cell lines.Condensin complexes regulate mitotic progression and interphase chromatin structure in embryonic stem cells.BRCA1 haploinsufficiency leads to altered expression of genes involved in cellular proliferation and development.A macrohistone variant links dynamic chromatin compaction to BRCA1-dependent genome maintenance.Defining the genomic signature of the parous breast.RAP80-directed tuning of BRCA1 homologous recombination function at ionizing radiation-induced nuclear foci.Dysregulation of X chromosome inactivation in high grade ovarian serous adenocarcinomaInactivation of X-linked tumor suppressor genes in human cancer.The inactive X chromosome is epigenetically unstable and transcriptionally labile in breast cancerExpression and function of a large non-coding RNA gene XIST in human cancer.The lesser known story of X chromosome reactivation: a closer look into the reprogramming of the inactive X chromosome.Mechanisms of BRCA1 tumor suppressionImpact of intertumoral heterogeneity on predicting chemotherapy response of BRCA1-deficient mammary tumors.New twists in X-chromosome inactivation.BRCA1-hapoinsufficiency: Unraveling the molecular and cellular basis for tissue-specific cancerReactivation of the inactive X chromosome in development and reprogrammingGuided by RNAs: X-inactivation as a model for lncRNA function.Misbehaviour of XIST RNA in breast cancer cells.Telomeric allelic imbalance indicates defective DNA repair and sensitivity to DNA-damaging agents.BRCA1-deficient mammary tumor cells are dependent on EZH2 expression and sensitive to Polycomb Repressive Complex 2-inhibitor 3-deazaneplanocin A.Increased expression of long non-coding RNA XIST predicts favorable prognosis of cervical squamous cell carcinoma subsequent to definitive chemoradiation therapy.Secretome proteomics reveals candidate non-invasive biomarkers of BRCA1 deficiency in breast cancer.Homozygosity for the WRN Helicase-Inactivating Variant, R834C, does not confer a Werner syndrome clinical phenotype.Epithelial-to-mesenchymal transition: possible role in meningiomas.Familial skewed X-chromosome inactivation linked to a component of the cohesin complex, SA2.Deciphering the roles of lncRNAs in breast development and disease.Long non-coding RNAs in ovarian cancer.
P2860
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P2860
Further evidence for BRCA1 communication with the inactive X chromosome.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Further evidence for BRCA1 communication with the inactive X chromosome.
@en
Further evidence for BRCA1 communication with the inactive X chromosome.
@nl
type
label
Further evidence for BRCA1 communication with the inactive X chromosome.
@en
Further evidence for BRCA1 communication with the inactive X chromosome.
@nl
prefLabel
Further evidence for BRCA1 communication with the inactive X chromosome.
@en
Further evidence for BRCA1 communication with the inactive X chromosome.
@nl
P2093
P50
P1433
P1476
Further evidence for BRCA1 communication with the inactive X chromosome.
@en
P2093
Daniel P Silver
David M Livingston
Elena Shestakova
Jean Feunteun
Jos Jonkers
Nicholas Denunzio
Rebecca Gelman
Serban Dragomir
Shihua D Lu
Shridar Ganesan
P304
P356
10.1016/J.CELL.2007.02.025
P407
P577
2007-03-01T00:00:00Z