Loss of LAP2 alpha delays satellite cell differentiation and affects postnatal fiber-type determination.
about
Lamina-associated polypeptide (LAP)2α and other LEM proteins in cancer biologyPhysiological and Pathological Aging Affects Chromatin Dynamics, Structure and Function at the Nuclear EdgeEmbryonic senescence and laminopathies in a progeroid zebrafish modelWhole exome sequencing identifies a novel EMD mutation in a Chinese family with dilated cardiomyopathyNetworking in the nucleus: a spotlight on LEM-domain proteinsExpression of Leukemia-Associated Nup98 Fusion Proteins Generates an Aberrant Nuclear Envelope Phenotype.Mechanics of the nucleus.Proliferation of progeria cells is enhanced by lamina-associated polypeptide 2α (LAP2α) through expression of extracellular matrix proteinsMuscle development, regeneration and laminopathies: how lamins or lamina-associated proteins can contribute to muscle development, regeneration and disease.Defective skeletal muscle growth in lamin A/C-deficient mice is rescued by loss of Lap2α.The search for evolutionary developmental origins of aging in zebrafish: a novel intersection of developmental and senescence biology in the zebrafish model system.Lamina-associated polypeptide (LAP)2α and nucleoplasmic lamins in adult stem cell regulation and disease.Lamins at the crossroads of mechanosignalingLamins in the nuclear interior - life outside the lamina.A-type lamins bind both hetero- and euchromatin, the latter being regulated by lamina-associated polypeptide 2 alpha.The muscle dystrophy-causing ΔK32 lamin A/C mutant does not impair the functions of the nucleoplasmic lamin-A/C-LAP2α complex in mice.Nucleoplasmic Lamin A/C and Polycomb group of proteins: An evolutionarily conserved interplay.Prepatterning of differentiation-driven nuclear lamin A/C-associated chromatin domains by GlcNAcylated histone H2B.Multiple novel functions of lamina associated polypeptide 2α in striated musclePrelamin A-mediated recruitment of SUN1 to the nuclear envelope directs nuclear positioning in human muscle.Lamina-associated polypeptide 1 is dispensable for embryonic myogenesis but required for postnatal skeletal muscle growth.Laminopathy-causing lamin A mutations reconfigure lamina-associated domains and local spatial chromatin conformation.
P2860
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P2860
Loss of LAP2 alpha delays satellite cell differentiation and affects postnatal fiber-type determination.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh-hant
name
Loss of LAP2 alpha delays sate ...... atal fiber-type determination.
@en
Loss of LAP2 alpha delays sate ...... atal fiber-type determination.
@nl
type
label
Loss of LAP2 alpha delays sate ...... atal fiber-type determination.
@en
Loss of LAP2 alpha delays sate ...... atal fiber-type determination.
@nl
prefLabel
Loss of LAP2 alpha delays sate ...... atal fiber-type determination.
@en
Loss of LAP2 alpha delays sate ...... atal fiber-type determination.
@nl
P2093
P356
P1433
P1476
Loss of LAP2 alpha delays sate ...... atal fiber-type determination.
@en
P2093
Colin L Stewart
Ivana Gotic
Juliane Braun
Katarzyna Biadasiewicz
Michael Leschnik
Rita Spilka
Wolfgang M Schmidt
P304
P356
10.1002/STEM.292
P577
2010-03-01T00:00:00Z