Extracellular matrix: The driving force of mammalian diseases.

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Extracellular matrix: The driving force of mammalian diseases.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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Extracellular matrix: The driving force of mammalian diseases.

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Extracellular matrix: The driving force of mammalian diseases.

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Extracellular matrix: The driving force of mammalian diseases.

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Extracellular matrix: The driving force of mammalian diseases.

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Extracellular matrix: The driving force of mammalian diseases.

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Extracellular matrix: The driving force of mammalian diseases.

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P1476

Extracellular matrix: The driving force of mammalian diseases.

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P2093

Maria A Gubbiotti

http://www.w3.org/2001/XMLSchema#string

Renato V Iozzo

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P2860

Proteoglycan form and function: A comprehensive nomenclature of proteoglycans

Decorin causes autophagy in endothelial cells via Peg3

Endorepellin, the angiostatic module of perlecan, interacts with both the α2β1 integrin and vascular endothelial growth factor receptor 2 (VEGFR2): a dual receptor antagonism

The putative tumour suppressor EXT1 alters the expression of cell-surface heparan sulfate

Decorin activates AMPK, an energy sensor kinase, to induce autophagy in endothelial cells

Perlecan maintains the integrity of cartilage and some basement membranes

The zebrafish candyfloss mutant implicates extracellular matrix adhesion failure in laminin alpha2-deficient congenital muscular dystrophy.

Inflammation, fibrosis, and modulation of the process by mesenchymal stem/stromal cells

Metalloproteinases: A parade of functions in matrix biology and an outlook for the future

Perlecan is essential for cartilage and cephalic development

P31

scholarly article

P356

10.1016/J.MATBIO.2018.03.023

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P577

2018-04-03T00:00:00Z

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P698

29625183

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P932

6146050

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