about
Effects of Long-Term Environmental Enrichment on Anxiety, Memory, Hippocampal Plasticity and Overall Brain Gene Expression in C57BL6 Mice.Intraneuronal pyroglutamate-Abeta 3-42 triggers neurodegeneration and lethal neurological deficits in a transgenic mouse modelPyroglutamate amyloid β (Aβ) aggravates behavioral deficits in transgenic amyloid mouse model for Alzheimer disease.Focusing the amyloid cascade hypothesis on N-truncated Abeta peptides as drug targets against Alzheimer's diseaseAxonal degeneration in an Alzheimer mouse model is PS1 gene dose dependent and linked to intraneuronal Aβ accumulation.Traumatic brain injury: cause or risk of Alzheimer's disease? A review of experimental studies.Neuron loss in transgenic mouse models of Alzheimer's diseaseThe Arctic AβPP mutation leads to Alzheimer's disease pathology with highly variable topographic deposition of differentially truncated AβEffect of copper intake on CSF parameters in patients with mild Alzheimer's disease: a pilot phase 2 clinical trial.Deposition of C-terminally truncated Aβ species Aβ37 and Aβ39 in Alzheimer's disease and transgenic mouse models.Motor impairment in Alzheimer's disease and transgenic Alzheimer's disease mouse models.Physical activity delays hippocampal neurodegeneration and rescues memory deficits in an Alzheimer disease mouse modelN-truncated Abeta starting with position four: early intraneuronal accumulation and rescue of toxicity using NT4X-167, a novel monoclonal antibody.Deciphering the molecular profile of plaques, memory decline and neuron loss in two mouse models for Alzheimer's disease by deep sequencing.Intraneuronal Aβ accumulation and neurodegeneration: lessons from transgenic models.Phosphorylation of the amyloid β-peptide at Ser26 stabilizes oligomeric assembly and increases neurotoxicity.Limited Effects of Prolonged Environmental Enrichment on the Pathology of 5XFAD Mice.Altered cholesterol metabolism in APP695-transfected neuroblastoma cells.OTX1 and OTX2 expression correlates with the clinicopathologic classification of medulloblastomas.N-truncated Aβ4-x peptides in sporadic Alzheimer's disease cases and transgenic Alzheimer mouse models.Gene expression of neuregulin-1 isoforms in different brain regions of elderly schizophrenia patients.Concomitant detection of beta-amyloid peptides with N-terminal truncation and different C-terminal endings in cortical plaques from cases with Alzheimer's disease, senile monkeys and triple transgenic mice.N-truncated Aβ2-X starting with position two in sporadic Alzheimer's disease cases and two Alzheimer mouse models.Decreased plasma cholesterol levels during aging in transgenic mouse models of Alzheimer's disease.Gene Dosage Dependent Aggravation of the Neurological Phenotype in the 5XFAD Mouse Model of Alzheimer's Disease.Age-dependent axonal degeneration in an Alzheimer mouse model.Neprilysin deficiency alters the neuropathological and behavioral phenotype in the 5XFAD mouse model of Alzheimer's disease.Intraneuronal APP/A beta trafficking and plaque formation in beta-amyloid precursor protein and presenilin-1 transgenic mice.I716F AβPP mutation associates with the deposition of oligomeric pyroglutamate amyloid-β and α-synucleinopathy with Lewy bodies.Antibody 9D5 recognizes oligomeric pyroglutamate amyloid-β in a fraction of amyloid-β deposits in Alzheimer's disease without cross-reactivity with other protein aggregates.Gene Expression Profiling in the APP/PS1KI Mouse Model of Familial Alzheimer's Disease.Synergistic Effect on Neurodegeneration by N-Truncated Aβ4-42 and Pyroglutamate Aβ3-42 in a Mouse Model of Alzheimer's Disease.Altered neurogenesis in mouse models of Alzheimer disease.Amyloid precursor protein is a biomarker for transformed human pluripotent stem cells.AβPP accumulation and/or intraneuronal amyloid-β accumulation? The 3xTg-AD mouse model revisited.Endogenous Apolipoprotein E (ApoE) Fragmentation Is Linked to Amyloid Pathology in Transgenic Mouse Models of Alzheimer's Disease.Lewy body variant of Alzheimer's disease: alpha-synuclein in dystrophic neurites of A beta plaques.Axonopathy in an APP/PS1 transgenic mouse model of Alzheimer's disease.Intracellular Aß triggers neuron loss in the cholinergic system of the APP/PS1KI mouse model of Alzheimer's disease.[Alzheimer's disease. Molecular pathology, animal models, and current treatment].
P50
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P50
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hulumtues
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Oliver Wirths
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Oliver Wirths
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Oliver Wirths
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Oliver Wirths
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Oliver Wirths
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Oliver Wirths
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Oliver Wirths
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P106
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P2798
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P3835
oliver-wirths2
P496
0000-0002-4115-0334