High-risk human papillomavirus E7 expression reduces cell-surface MHC class I molecules and increases susceptibility to natural killer cells.
about
Low NKp30, NKp46 and NKG2D expression and reduced cytotoxic activity on NK cells in cervical cancer and precursor lesions.Human papillomavirus 16 E5 modulates the expression of host microRNAsNK cells and virus-related cancers.An RNA aptamer provides a novel approach for the induction of apoptosis by targeting the HPV16 E7 oncoprotein.HLA class I and II expression in oropharyngeal squamous cell carcinoma in relation to tumor HPV status and clinical outcomeThe immune response to papillomavirus during infection persistence and regression.Viral proteins interfering with antigen presentation target the major histocompatibility complex class I peptide-loading complex.Human papillomavirus molecular biology and disease association.Human papillomavirus, high-grade intraepithelial neoplasia and killer immunoglogulin-like receptors: a Western Australian cohort study.The immune response in HPV(+) oropharyngeal cancer.Meta-analysis of survival in patients with HNSCC discriminates risk depending on combined HPV and p16 status.Human papillomavirus-driven immune deviation: challenge and novel opportunity for immunotherapy.The human papillomavirus E7 oncoprotein as a regulator of transcription.Analysis of Class I Major Histocompatibility Complex Gene Transcription in Human Tumors Caused by Human Papillomavirus Infection.Identification of key amino acid residues that determine the ability of high risk HPV16-E7 to dysregulate major histocompatibility complex class I expression.MHC class I expression in HPV positive and negative tonsillar squamous cell carcinoma in correlation to clinical outcome.Microparticles produced by human papillomavirus type 16 E7-expressing cells impair antigen presenting cell function and the cytotoxic T cell response.Human papillomavirus entry into NK cells requires CD16 expression and triggers cytotoxic activity and cytokine secretion
P2860
Q33468660-1145B860-DDA2-4D90-8688-B3915D8B3D5DQ33958050-C2A72A00-AF04-4C87-94A2-6B5E95DD06D2Q34337237-F8D08DF6-A528-4712-A7BB-791A60C1B622Q34758890-F01DB419-A9B1-4972-9686-D412F2597ACBQ35018702-990F8D89-1654-4BB2-9727-A68984AD9806Q36540399-DBC7768D-8FF3-4D7F-BFC6-A3191B85406AQ37151597-7D0A0E58-EAB5-4997-ABE8-F56441A17259Q37257349-4240200B-D105-4B0C-9800-333FCD2DD1B6Q37356179-67B17765-3932-46EB-84C1-A8D20FFB297FQ37562953-5AEF3473-093C-428E-978E-A6D7DE7FDE54Q38557897-CE4CF94F-BE22-43DC-818E-9D0988188A81Q38644639-B7439233-6A67-410B-AB55-02A3C322C459Q39000409-FBB90104-8071-4C12-954E-33007CC97AEFQ40047932-73748919-59C8-496F-B438-3F8698152F78Q42030463-AA3F6AD4-74DB-4B9D-9578-4557DA643966Q43462890-E1FE055E-F2A8-4108-9D7C-7E92D0DE6007Q48102982-3313769B-51C3-4D78-81C5-6C5569AA6809Q57580316-8C2D1F2B-65D4-4810-BF35-020C1974E3E0
P2860
High-risk human papillomavirus E7 expression reduces cell-surface MHC class I molecules and increases susceptibility to natural killer cells.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
High-risk human papillomavirus ...... ility to natural killer cells.
@en
High-risk human papillomavirus ...... ility to natural killer cells.
@en-gb
High-risk human papillomavirus ...... ility to natural killer cells.
@nl
type
label
High-risk human papillomavirus ...... ility to natural killer cells.
@en
High-risk human papillomavirus ...... ility to natural killer cells.
@en-gb
High-risk human papillomavirus ...... ility to natural killer cells.
@nl
prefLabel
High-risk human papillomavirus ...... ility to natural killer cells.
@en
High-risk human papillomavirus ...... ility to natural killer cells.
@en-gb
High-risk human papillomavirus ...... ility to natural killer cells.
@nl
P2093
P2860
P356
P1433
P1476
High-risk human papillomavirus ...... bility to natural killer cells
@en
P2093
P2860
P2888
P304
P356
10.1038/SJ.ONC.1210798
P407
P577
2007-09-10T00:00:00Z