about
Galectin-3 impairment of MYCN-dependent apoptosis-sensitive phenotype is antagonized by nutlin-3 in neuroblastoma cellsMolecular mechanisms of MYCN-dependent apoptosis and the MDM2-p53 pathway: an Achille's heel to be exploited for the therapy of MYCN-amplified neuroblastoma.Human papilloma virus-dependent HMGA1 expression is a relevant step in cervical carcinogenesisGalectin-3 is a marker of favorable prognosis and a biologically relevant molecule in neuroblastic tumors.Inhibition of STAT3 with the Generation 2.5 Antisense Oligonucleotide, AZD9150, Decreases Neuroblastoma Tumorigenicity and Increases Chemosensitivity.MYCN sensitizes human neuroblastoma to apoptosis by HIPK2 activation through a DNA damage response.The MRN complex is transcriptionally regulated by MYCN during neural cell proliferation to control replication stress.CRISPR-Cas9 screen reveals a MYCN-amplified neuroblastoma dependency on EZH2.Epigenetic siRNA and Chemical Screens Identify SETD8 Inhibition as a Therapeutic Strategy for p53 Activation in High-Risk Neuroblastoma.The HMGA1 protoncogene frequently deregulated in cancer is a transcriptional target of E2F1.
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description
hulumtuese
@sq
researcher
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wetenschapper
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հետազոտող
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name
Veronica Veschi
@ast
Veronica Veschi
@en
Veronica Veschi
@es
Veronica Veschi
@nl
Veronica Veschi
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type
label
Veronica Veschi
@ast
Veronica Veschi
@en
Veronica Veschi
@es
Veronica Veschi
@nl
Veronica Veschi
@sl
prefLabel
Veronica Veschi
@ast
Veronica Veschi
@en
Veronica Veschi
@es
Veronica Veschi
@nl
Veronica Veschi
@sl
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P1153
24482692900
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P496
0000-0002-6379-7366