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A review of Brucella infection in marine mammals, with special emphasis on Brucella pinnipedialis in the hooded seal (Cystophora cristata)Type IV secretion system of Brucella spp. and its effectorsBrucella induces an unfolded protein response via TcpB that supports intracellular replication in macrophagesBrucella abortus uses a stealthy strategy to avoid activation of the innate immune system during the onset of infection.Brucella control of dendritic cell maturation is dependent on the TIR-containing protein Btp1.RNAi screen of endoplasmic reticulum-associated host factors reveals a role for IRE1alpha in supporting Brucella replication.DNA polymorphism analysis of Brucella lipopolysaccharide genes reveals marked differences in O-polysaccharide biosynthetic genes between smooth and rough Brucella species and novel species-specific markers.The differential interaction of Brucella and ochrobactrum with innate immunity reveals traits related to the evolution of stealthy pathogensCaspase-2 mediated apoptotic and necrotic murine macrophage cell death induced by rough Brucella abortusDiscordant Brucella melitensis antigens yield cognate CD8+ T cells in vivo.The Brucella abortus phosphoglycerate kinase mutant is highly attenuated and induces protection superior to that of vaccine strain 19 in immunocompromised and immunocompetent miceMechanism of Asp24 upregulation in Brucella abortus rough mutant with a disrupted O-antigen export system and effect of Asp24 in bacterial intracellular survival.Mutants in the lipopolysaccharide of Brucella ovis are attenuated and protect against B. ovis infection in miceEndocytosis of viruses and bacteria.Antigenic, immunologic and genetic characterization of rough strains B. abortus RB51, B. melitensis B115 and B. melitensis B18.The lipopolysaccharide core of Brucella abortus acts as a shield against innate immunity recognitionThe AS87_04050 gene is involved in bacterial lipopolysaccharide biosynthesis and pathogenicity of Riemerella anatipestiferMicroRNA expression profile in RAW264.7 cells in response to Brucella melitensis infectionCaspase-2-dependent dendritic cell death, maturation, and priming of T cells in response to Brucella abortus infection.Comparative analysis of the early transcriptome of Brucella abortus--infected monocyte-derived macrophages from cattle naturally resistant or susceptible to brucellosisBlocking the expression of syntaxin 4 interferes with initial phagocytosis of Brucella melitensis in macrophagesEstablishment of systemic Brucella melitensis infection through the digestive tract requires urease, the type IV secretion system, and lipopolysaccharide O antigenBrucella abortus Induces the Premature Death of Human Neutrophils through the Action of Its Lipopolysaccharide.Quorum-sensing and BvrR/BvrS regulation, the type IV secretion system, cyclic glucans, and BacA in the virulence of Brucella ovis: similarities to and differences from smooth brucellaeTranscriptome analysis of HeLa cells response to Brucella melitensis infection: a molecular approach to understand the role of the mucosal epithelium in the onset of the Brucella pathogenesis.BvrR/BvrS-controlled outer membrane proteins Omp3a and Omp3b are not essential for Brucella abortus virulence.CCL20 and Beta-Defensin 2 Production by Human Lung Epithelial Cells and Macrophages in Response to Brucella abortus Infection.Characterization of recombinant B. abortus strain RB51SOD toward understanding the uncorrelated innate and adaptive immune responses induced by RB51SOD compared to its parent vaccine strain RB51Antigen-specific acquired immunity in human brucellosis: implications for diagnosis, prognosis, and vaccine development.Comparative genomics of early-diverging Brucella strains reveals a novel lipopolysaccharide biosynthesis pathway.Pathogen-endoplasmic-reticulum interactions: in through the out door.Smooth to Rough Dissociation in Brucella: The Missing Link to VirulenceBrucella spp noncanonical LPS: structure, biosynthesis, and interaction with host immune system.Defensin susceptibility and colonization in the mouse model of AJ100, a polymyxin B-resistant, Brucella abortus RB51 isolate.The study of the core part and non-repeating elements of the O-antigen of Brucella lipopolysaccharideComparison of Biological and Immunological Characterization of Lipopolysaccharides From Brucella abortus RB51 and S19.Vaccination of Elk (Cervus canadensis) with Brucella abortus Strain RB51 Overexpressing Superoxide Dismutase and Glycosyltransferase Genes Does Not Induce Adequate Protection against Experimental Brucella abortus Challenge.Structural Studies of Lipopolysaccharide-defective Mutants from Brucella melitensis Identify a Core Oligosaccharide Critical in Virulence.Genomic island 2 of Brucella melitensis is a major virulence determinant: functional analyses of genomic islands.Brucella discriminates between mouse dendritic cell subsets upon in vitro infection.
P2860
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P2860
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Brucella lipopolysaccharide acts as a virulence factor.
@en
Brucella lipopolysaccharide acts as a virulence factor.
@nl
type
label
Brucella lipopolysaccharide acts as a virulence factor.
@en
Brucella lipopolysaccharide acts as a virulence factor.
@nl
prefLabel
Brucella lipopolysaccharide acts as a virulence factor.
@en
Brucella lipopolysaccharide acts as a virulence factor.
@nl
P50
P1476
Brucella lipopolysaccharide acts as a virulence factor.
@en
P2093
Edgardo Moreno
P356
10.1016/J.MIB.2004.12.003
P577
2005-02-01T00:00:00Z