about
Mal (MyD88-adapter-like) is required for Toll-like receptor-4 signal transductionToll-like receptor mRNA expression is selectively increased in the colonic mucosa of two animal models relevant to irritable bowel syndromeMicrobiota-host interactions in irritable bowel syndrome: epithelial barrier, immune regulation and brain-gut interactionsCharacterization of signaling pathways activated by the interleukin 1 (IL-1) receptor homologue T1/ST2. A role for Jun N-terminal kinase in IL-4 induction.ST2 is an inhibitor of interleukin 1 receptor and Toll-like receptor 4 signaling and maintains endotoxin tolerance.Crystal structure of the Toll/interleukin-1 receptor domain of human IL-1RAPL.Engagement of Fas on Macrophages Modulates Poly I:C induced cytokine production with specific enhancement of IP-10.An antitumorigenic role for the IL-33 receptor, ST2L, in colon cancer.Tracing innate immune defences along the path of Listeria monocytogenes infection.Differential expression of key regulators of Toll-like receptors in ulcerative colitis and Crohn's disease: a role for Tollip and peroxisome proliferator-activated receptor gamma?Wounds that heal and wounds that don't - The role of the IL-33/ST2 pathway in tissue repair and tumorigenesis.Intestinal expression of Fas and Fas ligand is upregulated by bacterial signaling through TLR4 and TLR5, with activation of Fas modulating intestinal TLR-mediated inflammation.Identification of TLR10 as a key mediator of the inflammatory response to Listeria monocytogenes in intestinal epithelial cells and macrophages.IL-36α expression is elevated in ulcerative colitis and promotes colonic inflammation.Mal interacts with tumor necrosis factor receptor-associated factor (TRAF)-6 to mediate NF-kappaB activation by toll-like receptor (TLR)-2 and TLR4.Bruton's tyrosine kinase is a Toll/interleukin-1 receptor domain-binding protein that participates in nuclear factor kappaB activation by Toll-like receptor 4.Bcl-3 deficiency protects against dextran-sodium sulphate-induced colitis in the mouse.Differential expression of toll-like receptors in patients with irritable bowel syndrome.Engagement of Fas differentially regulates the production of LPS-induced proinflammatory cytokines and type I interferons
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Elizabeth K Brint
@ast
Elizabeth K Brint
@en
Elizabeth K Brint
@es
Elizabeth K Brint
@nl
Elizabeth K Brint
@sl
type
label
Elizabeth K Brint
@ast
Elizabeth K Brint
@en
Elizabeth K Brint
@es
Elizabeth K Brint
@nl
Elizabeth K Brint
@sl
prefLabel
Elizabeth K Brint
@ast
Elizabeth K Brint
@en
Elizabeth K Brint
@es
Elizabeth K Brint
@nl
Elizabeth K Brint
@sl
P106
P21
P31
P496
0000-0001-9214-149X