Alternative mRNA Splicing in the Pathogenesis of Obesity.

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Alternative mRNA Splicing in the Pathogenesis of Obesity.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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2018年學術文章

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name

Alternative mRNA Splicing in the Pathogenesis of Obesity.

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Alternative mRNA Splicing in the Pathogenesis of Obesity.

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Item

label

Alternative mRNA Splicing in the Pathogenesis of Obesity.

@en

Alternative mRNA Splicing in the Pathogenesis of Obesity.

@nl

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Alternative mRNA Splicing in the Pathogenesis of Obesity.

@en

Alternative mRNA Splicing in the Pathogenesis of Obesity.

@nl

P1476

Alternative mRNA Splicing in the Pathogenesis of Obesity

@en

P2093

Lu Xu

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Mabel Yin-Chun Yau

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P275

Creative Commons Attribution

P2860

Adipocyte NCoR knockout decreases PPARγ phosphorylation and enhances PPARγ activity and insulin sensitivity

Structural organization of the human gene encoding nuclear lamin A and nuclear lamin C

A mutation in the human leptin receptor gene causes obesity and pituitary dysfunction

Cell signaling by receptor tyrosine kinases

Modulating splicing with small molecular inhibitors of the spliceosome

Brown Fat and Browning for the Treatment of Obesity and Related Metabolic Disorders

Introns: The Functional Benefits of Introns in Genomes

The Role of Alternative Splicing in the Control of Immune Homeostasis and Cellular Differentiation

Is spinal muscular atrophy a disease of the motor neurons only: pathogenesis and therapeutic implications?

Mouse models of laminopathies

P31

scholarly article

P356

10.3390/IJMS19020632

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P407

English

P433

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P478

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P50

Chi-Ming Wong

P5008

ScienceSource

P577

2018-02-23T00:00:00Z

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P698

29473878

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P921

Adipositas

regulation of gene expression

P932

5855854

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