about
PGC-1alpha deficiency causes multi-system energy metabolic derangements: muscle dysfunction, abnormal weight control and hepatic steatosisMetabolic determinants of electrical failure in ex-vivo canine model of cardiac arrest: evidence for the protective role of inorganic pyrophosphateAberrant water homeostasis detected by stable isotope analysisLoss of bradykinin signaling does not accelerate the development of cardiac dysfunction in type 1 diabetic akita mice.PGC-1alpha coactivates PDK4 gene expression via the orphan nuclear receptor ERRalpha: a mechanism for transcriptional control of muscle glucose metabolismKnockout of insulin receptors in cardiomyocytes attenuates coronary arterial dysfunction induced by pressure overloadCytosolic, but not mitochondrial, oxidative stress is a likely contributor to cardiac hypertrophy resulting from cardiac specific GLUT4 deletion in miceGenetic loss of insulin receptors worsens cardiac efficiency in diabetes.A conserved role for phosphatidylinositol 3-kinase but not Akt signaling in mitochondrial adaptations that accompany physiological cardiac hypertrophy.Cardiac PI3K-Akt impairs insulin-stimulated glucose uptake independent of mTORC1 and GLUT4 translocation.Regulation of fatty acid metabolism by mTOR in adult murine hearts occurs independently of changes in PGC-1αMy Sweetheart Is Broken: Role of Glucose in Diabetic Cardiomyopathy.Diabetes-Related Cardiac Dysfunction.Glucose transporter 4-deficient hearts develop maladaptive hypertrophy in response to physiological or pathological stresses.Calcineurin and calcium/calmodulin-dependent protein kinase activate distinct metabolic gene regulatory programs in cardiac muscleGenome-wide DNA methylation encodes cardiac transcriptional reprogramming in human ischemic heart failure.Cardiac hypertrophy caused by peroxisome proliferator- activated receptor-gamma agonist treatment occurs independently of changes in myocardial insulin signalingThe maximal downstroke of epicardial potentials as an index of electrical activity in mouse heartsAbsence of glucose transporter 4 diminishes electrical activity of mouse hearts during hypoxiaMitoQ regulates redox-related noncoding RNAs to preserve mitochondrial network integrity in pressure-overload heart failureNovel role of the ER/SR Ca2+ sensor STIM1 in the regulation of cardiac metabolismDNA methylation reprograms cardiac metabolic gene expression in end-stage human heart failure
P50
Q24798075-9393EA00-1F3F-4396-8503-ECE3969E089DQ27312320-A7B07A86-2CB6-412D-A4BA-40F1ABE9BDA5Q28750261-A1AC4427-5910-4E4B-A383-EA158AFA0D2EQ34132504-D22A4250-F5D8-4F2F-A11F-D78F82BA2F0DQ34230877-FD927AF3-DD52-4076-BA02-3EC96B30E9FFQ34502029-0BE3087C-0840-47A1-A432-117086237B93Q35700338-1B4830B3-9B2E-451B-8329-23914EF5633EQ35892530-403C9B61-201C-420D-9D3F-6431E5FD5E3FQ36156412-B303381D-F0FD-4C8F-A616-E4AF31F5F27CQ36533391-4DF3BC25-4DD6-422D-9272-5CDFA679447BQ37054943-72AE8E65-BB64-476A-B9E6-AA3FAFC61970Q37670082-6420CD2A-AC3F-4CAD-9E33-2419D7E1F493Q39420123-2129C25A-2692-444A-A800-245E3D602D49Q47931224-9613EF30-3020-40E5-8634-3F04CA7C6910Q64379442-B3594BA9-8303-41D4-84A8-5FF7DB6F789BQ64891344-FFE4FB42-5898-4803-BF97-209C39BA4CDAQ81152659-2D2DAFF7-B3DE-4BDF-83A4-D1BB4E88E35AQ84806337-BED3F27C-3D2A-4EBA-A502-2A418F73D538Q85528907-14460B37-53C4-460A-B080-C4A91B328647Q89466157-95AFB6CC-E41C-45C3-8A1E-8D3A6971BB47Q90692354-7F1B2214-2039-4CF9-A17D-4C5948D24C82Q91823131-20D53A9A-2C18-44C0-B412-5AFF23BF5E97
P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Adam R. Wende
@ast
Adam R. Wende
@en
Adam R. Wende
@es
Adam R. Wende
@nl
Adam R. Wende
@sl
type
label
Adam R. Wende
@ast
Adam R. Wende
@en
Adam R. Wende
@es
Adam R. Wende
@nl
Adam R. Wende
@sl
prefLabel
Adam R. Wende
@ast
Adam R. Wende
@en
Adam R. Wende
@es
Adam R. Wende
@nl
Adam R. Wende
@sl
P106
P1153
6602484414
P21
P31
P496
0000-0002-5536-4675