about
Trypanosoma brucei TbIF1 inhibits the essential F1-ATPase in the infectious form of the parasite.Secondary structure is required for 3' splice site recognition in yeast.The F1 -ATPase from Trypanosoma brucei is elaborated by three copies of an additional p18-subunit.Nineteen complex-related factor Prp45 is required for the early stages of cotranscriptional spliceosome assembly.ATP synthase from Trypanosoma brucei has an elaborated canonical F1-domain and conventional catalytic sites.Evaluation of the Trypanosoma brucei 6-oxopurine salvage pathway as a potential target for drug discovery.SKIP is a component of the spliceosome linking alternative splicing and the circadian clock in Arabidopsis.Inhibition of F -ATPase from Trypanosoma brucei by its regulatory protein inhibitor TbIFChromosome 21 gain is dispensable for transient myeloproliferative disorder driven by a novel GATA1 mutationIsolation of F1-ATPase from the Parasitic Protist Trypanosoma bruceiCauses and Effects of Loss of Classical Nonhomologous End Joining Pathway in Parasitic Eukaryotes
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Ondřej Gahura
@ast
Ondřej Gahura
@en
Ondřej Gahura
@es
Ondřej Gahura
@nl
Ondřej Gahura
@sl
type
label
Ondřej Gahura
@ast
Ondřej Gahura
@en
Ondřej Gahura
@es
Ondřej Gahura
@nl
Ondřej Gahura
@sl
prefLabel
Ondřej Gahura
@ast
Ondřej Gahura
@en
Ondřej Gahura
@es
Ondřej Gahura
@nl
Ondřej Gahura
@sl
P106
P1153
26021788500
P31
P496
0000-0002-2925-4763