Initial Influenza Virus Replication Can Be Limited in Allergic Asthma Through Rapid Induction of Type III Interferons in Respiratory Epithelium.

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Initial Influenza Virus Replication Can Be Limited in Allergic Asthma Through Rapid Induction of Type III Interferons in Respiratory Epithelium.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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Initial Influenza Virus Replic ...... ons in Respiratory Epithelium.

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Initial Influenza Virus Replic ...... ons in Respiratory Epithelium.

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Initial Influenza Virus Replic ...... ons in Respiratory Epithelium.

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Initial Influenza Virus Replic ...... ons in Respiratory Epithelium.

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Initial Influenza Virus Replic ...... ons in Respiratory Epithelium.

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Initial Influenza Virus Replic ...... ons in Respiratory Epithelium.

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P1476

Initial Influenza Virus Replic ...... rons in Respiratory Epithelium

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P2093

Ara Jo

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Hye Young Kim

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Hyun Jik Kim

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Hyun Jung Lim

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Sujin An

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Sung Woo Cho

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Young Eun Han

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P2860

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Interferon response and respiratory virus control are preserved in bronchial epithelial cells in asthma.

Prevention of Influenza Virus-Induced Immunopathology by TGF-β Produced during Allergic Asthma.

Mice with asthma are more resistant to influenza virus infection and NK cells activated by the induction of asthma have potentially protective effects.

Type 1 interferons and the virus-host relationship: a lesson in détente .

The regulation of allergy and asthma.

Rhinovirus-induced lower respiratory illness is increased in asthma and related to virus load and Th1/2 cytokine and IL-10 production.

Innate immunity in the pathogenesis of virus-induced asthma exacerbations.

Interactions between innate and adaptive immunity in asthma pathogenesis: new perspectives from studies on acute exacerbations.

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scholarly article

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10.3389/FIMMU.2018.00986

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Yung Jin Jeon

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2018-05-17T00:00:00Z

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29867963

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