about
Cryptococcus and Phagocytes: Complex Interactions that Influence Disease OutcomeVaccine-mediated immune responses to experimental pulmonary Cryptococcus gattii infection in miceContribution of cell surface hydrophobicity protein 1 (Csh1p) to virulence of hydrophobic Candida albicans serotype A cells.Insights into the mechanisms of protective immunity against Cryptococcus neoformans infection using a mouse model of pulmonary cryptococcosisPulmonary infection with an interferon-gamma-producing Cryptococcus neoformans strain results in classical macrophage activation and protection.Characterization of IL-22 and antimicrobial peptide production in mice protected against pulmonary Cryptococcus neoformans infection.Role of IL-17A on resolution of pulmonary C. neoformans infectionCandidacidal activity of synthetic peptides based on the antimicrobial domain of the neutrophil-derived protein, CAP37Candida-specific antibodies during experimental vaginal candidiasis in mice.STAT1 signaling is essential for protection against Cryptococcus neoformans infection in mice.Interleukin-17 is not required for classical macrophage activation in a pulmonary mouse model of Cryptococcus neoformans infection.The acute neutrophil response mediated by S100 alarmins during vaginal Candida infections is independent of the Th17-pathway.Depletion of neutrophils in a protective model of pulmonary cryptococcosis results in increased IL-17A production by γδ T cells.Isolation and purification of antigenic components of CryptococcusDevelopment of protective inflammation and cell-mediated immunity against Cryptococcus neoformans after exposure to hyphal mutants.Dectin-3 Is Not Required for Protection against Cryptococcus neoformans Infection.STAT1 signaling within macrophages is required for antifungal activity against Cryptococcus neoformansAntifungal Activity of Plasmacytoid Dendritic Cells against Cryptococcus neoformans In Vitro Requires Expression of Dectin-3 (CLEC4D) and Reactive Oxygen Species.Molecules at the interface of Cryptococcus and the host that determine disease susceptibility.Flow Cytometric Analysis of Protective T-Cell Response Against Pulmonary Coccidioides Infection.Induction of Broad-Spectrum Protective Immunity against Disparate Cryptococcus Serotypes.Inhibitory effects of whole and parotid saliva on immunomodulators.IFN-γ immune priming of macrophages in vivo induces prolonged STAT1 binding and protection against Cryptococcus neoformansInfluence of periodontal disease on Th1/Th2-type cytokines in saliva of HIV-positive individuals
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description
hulumtuese
@sq
onderzoeker
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researcher
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հետազոտող
@hy
name
Karen L. Wozniak
@ast
Karen L. Wozniak
@en
Karen L. Wozniak
@es
Karen L. Wozniak
@nl
Karen L. Wozniak
@sl
type
label
Karen L. Wozniak
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Karen L. Wozniak
@en
Karen L. Wozniak
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Karen L. Wozniak
@nl
Karen L. Wozniak
@sl
prefLabel
Karen L. Wozniak
@ast
Karen L. Wozniak
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Karen L. Wozniak
@es
Karen L. Wozniak
@nl
Karen L. Wozniak
@sl
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P1153
7103066851
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0000-0003-1798-2146