about
Dose-response met-RANTES treatment of experimental periodontitis: a narrow edge between the disease severity attenuation and infection control.Endocytosis of DNA-Hsp65 alters the pH of the late endosome/lysosome and interferes with antigen presentation.Protection against tuberculosis by a single intranasal administration of DNA-hsp65 vaccine complexed with cationic liposomes.Angiogenesis and lymphangiogenesis in the spectrum of leprosy and its reactional forms.The potential role of suppressors of cytokine signaling in the attenuation of inflammatory reaction and alveolar bone loss associated with apical periodontitisAntigen-presenting cells transfected with Hsp65 messenger RNA fail to treat experimental tuberculosis.Inhibition of the myotoxic activity of Bothrops jararacussu venom and its two major myotoxins, BthTX-I and BthTX-II, by the aqueous extract of Tabernaemontana catharinensis A. DC. (Apocynaceae).Strong and persistent microbial and inflammatory stimuli overcome the genetic predisposition to higher matrix metalloproteinase-1 (MMP-1) expression: a mechanistic explanation for the lack of association of MMP1-1607 single-nucleotide polymorphism gThe synergy between structural stability and DNA-binding controls the antibody production in EPC/DOTAP/DOPE liposomes and DOTAP/DOPE lipoplexes.Evidence supporting a protective role for th9 and th22 cytokines in human and experimental periapical lesions.Oral implant osseointegration model in C57Bl/6 mice: microtomographic, histological, histomorphometric and molecular characterization.Association of human T lymphotropic virus 1 amplification of periodontitis severity with altered cytokine expression in response to a standard periodontopathogen infection.Experimental periodontitis in mice selected for maximal or minimal inflammatory reactions: increased inflammatory immune responsiveness drives increased alveolar bone loss without enhancing the control of periodontal infectionThe broad effects of the functional IL-10 promoter-592 polymorphism: modulation of IL-10, TIMP-3, and OPG expression and their association with periodontal disease outcomeFunctional interferences in host inflammatory immune response by airway allergic inflammation restrain experimental periodontitis development in miceThe use of chronic gingivitis as reference status increases the power and odds of periodontitis genetic studies: a proposal based in the exposure concept and clearer resistance and susceptibility phenotypes definitionPeriodontitis and arthritis interaction in mice involves a shared hyper-inflammatory genotype and functional immunological interferences
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description
hulumtuese
@sq
onderzoeker
@nl
researcher
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հետազոտող
@hy
name
Ana Paula F Trombone
@ast
Ana Paula F Trombone
@en
Ana Paula F Trombone
@es
Ana Paula F Trombone
@nl
Ana Paula F Trombone
@sl
type
label
Ana Paula F Trombone
@ast
Ana Paula F Trombone
@en
Ana Paula F Trombone
@es
Ana Paula F Trombone
@nl
Ana Paula F Trombone
@sl
prefLabel
Ana Paula F Trombone
@ast
Ana Paula F Trombone
@en
Ana Paula F Trombone
@es
Ana Paula F Trombone
@nl
Ana Paula F Trombone
@sl
P106
P21
P31
P496
0000-0002-8102-6098