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Loss of the RNA-binding protein TACO1 causes late-onset mitochondrial dysfunction in miceMutation in MRPS34 compromises protein synthesis and causes mitochondrial dysfunctionA new methodology for assessment of the performance of heartbeat classification systemsDystrophic Cardiomyopathy-Potential Role of Calcium in Pathogenesis, Treatment and Novel TherapiesSecreted frizzled-related protein 4: an angiogenesis inhibitor.Impaired functional communication between the L-type calcium channel and mitochondria contributes to metabolic inhibition in the mdx heart.Glutathionylation of the L-type Ca2+ channel in oxidative stress-induced pathology of the heart.Decreased myocardial injury and improved contractility after administration of a peptide derived against the alpha-interacting domain of the L-type calcium channelCholine kinase β mutant mice exhibit reduced phosphocholine, elevated osteoclast activity, and low bone massProtein kinase C isozyme selective peptides--a current view of what they tell us about location and function of isozymes in the heart.Acute hypoxia differentially regulates K(+) channels. Implications with respect to cardiac arrhythmia.Reactive oxygen species in cardiac signalling: from mitochondria to plasma membrane ion channels.Evidence for redox sensing by a human cardiac calcium channel.Redox control of calcium channels: from mechanisms to therapeutic opportunities.Mechanosensitive channel of large conductance.What cardiologists should know about calcium ion channels and their regulation by reactive oxygen species.MscS, the bacterial mechanosensitive channel of small conductance.Myeloperoxidase-derived oxidants inhibit sarco/endoplasmic reticulum Ca2+-ATPase activity and perturb Ca2+ homeostasis in human coronary artery endothelial cells.The impact of non-severe burn injury on cardiac function and long-term cardiovascular pathologyThe effect of acute hypoxia on excitability in the heart and the L-type calcium channel as a therapeutic target.The L-type Ca(2+) channel as a therapeutic target in heart disease.The L-type Ca2+ channel: A mediator of hypertrophic cardiomyopathyTargeting calcium and the mitochondria in prevention of pathology in the heart.How does calcium regulate mitochondrial energetics in the heart? - new insights.How does the heart sense changes in oxygen tension: a role for ion channels?Deranged sodium to sudden death.The potential for nanotechnology to improve delivery of therapy to the acute ischemic heart.L-type calcium channel: Clarifying the "oxygen sensing hypothesis".Specific combinations of ion channel inhibitors reduce excessive Ca(2+) influx as a consequence of oxidative stress and increase neuronal and glial cell viability in vitro.Auto-regulation in the powerhouse.Prolonged glutamate excitotoxicity increases GluR1 immunoreactivity but decreases mRNA of GluR1 and associated regulatory proteins in dissociated rat retinae in vitro.Adult-onset obesity is triggered by impaired mitochondrial gene expression.Genistein increases the sensitivity of cardiac ion channels to beta-adrenergic receptor stimulation.The L-type Ca(2+) channel facilitates abnormal metabolic activity in the cTnI-G203S mouse model of hypertrophic cardiomyopathy.Qo site of mitochondrial complex III is the source of increased superoxide after transient exposure to hydrogen peroxide.Evidence of altered guinea pig ventricular cardiomyocyte protein expression and growth in response to a 5 min in vitro exposure to H(2)O(2).Evidence for regulation of mitochondrial function by the L-type Ca2+ channel in ventricular myocytes.Identification of a novel cAMP dependent protein kinase A phosphorylation site on the human cardiac calcium channel.The cardiac L-type calcium channel alpha subunit is a target for direct redox modification during oxidative stress-the role of cysteine residues in the alpha interacting domain.Nanoparticle-mediated dual delivery of an antioxidant and a peptide against the L-Type Ca2+ channel enables simultaneous reduction of cardiac ischemia-reperfusion injury.
P50
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P50
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հետազոտող
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Livia C Hool
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Livia C Hool
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Livia C Hool
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Livia C Hool
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Livia C Hool
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Livia C Hool
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Livia C Hool
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Livia C Hool
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Livia C Hool
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Livia C Hool
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P106
P1153
6602116492
P31
P496
0000-0001-7758-5252