about
Chronic cocaine-regulated epigenomic changes in mouse nucleus accumbens.Kalirin regulates cortical spine morphogenesis and disease-related behavioral phenotypesKalirin-7 controls activity-dependent structural and functional plasticity of dendritic spinesACF chromatin-remodeling complex mediates stress-induced depressive-like behaviorEpac2 induces synapse remodeling and depression and its disease-associated forms alter spinesHippocampal phenotypes in kalirin-deficient mice.Kalirin loss results in cortical morphological alterationsA sequence variant in human KALRN impairs protein function and coincides with reduced cortical thickness.Control of interneuron dendritic growth through NRG1/erbB4-mediated kalirin-7 disinhibitionEpigenetic basis of opiate suppression of Bdnf gene expression in the ventral tegmental areaPathogenesis of hepatic encephalopathy in acute liver failure.Increased expression of Kalirin-9 in the auditory cortex of schizophrenia subjects: its role in dendritic pathologyRole of Tet1 and 5-hydroxymethylcytosine in cocaine action.Afadin is required for maintenance of dendritic structure and excitatory tone.Mefloquine in the nucleus accumbens promotes social avoidance and anxiety-like behavior in mice.Rac1 is essential in cocaine-induced structural plasticity of nucleus accumbens neuronsEpigenetic regulation of RAC1 induces synaptic remodeling in stress disorders and depressionKalirin-7 mediates cocaine-induced AMPA receptor and spine plasticity, enabling incentive sensitization.Neuregulin1 signaling promotes dendritic spine growth through kalirin.Histone arginine methylation in cocaine action in the nucleus accumbens.Convergent CaMK and RacGEF signals control dendritic structure and function.Coordination of synaptic adhesion with dendritic spine remodeling by AF-6 and kalirin-7.Dopaminergic dynamics underlying sex-specific cocaine reward.Prefrontal cortical circuit for depression- and anxiety-related behaviors mediated by cholecystokinin: role of ΔFosB.Bidirectional Synaptic Structural Plasticity after Chronic Cocaine Administration Occurs through Rap1 Small GTPase Signaling.WAVE1 in neurons expressing the D1 dopamine receptor regulates cellular and behavioral actions of cocainePhosphorylation and cleavage of tau in non-AD tauopathies.Tau truncation during neurofibrillary tangle evolution in Alzheimer's disease.Sex-specific transcriptional signatures in human depression.Pseudophosphorylation of tau at serine 422 inhibits caspase cleavage: in vitro evidence and implications for tangle formation in vivo.The dendritic spine morphogenic effects of repeated cocaine use occur through the regulation of serum response factor signaling.Estrogen receptor α drives pro-resilient transcription in mouse models of depression.Voluntary wheel running promotes resilience to chronic social defeat stress in mice: a role for nucleus accumbens ΔFosBKalirin-7 prevents dendritic spine dysgenesis induced by amyloid beta-derived oligomersWithdrawal from repeated morphine administration augments expression of the RhoA network in the nucleus accumbens to control synaptic structureSynaptic Microtubule-Associated Protein EB3 and SRC Phosphorylation Mediate Structural and Behavioral Adaptations During Withdrawal From Cocaine Self-AdministrationDysregulated Prefrontal Cortical RhoA Signal Transduction in Bipolar Disorder with Psychosis: New Implications for Disease Pathophysiology
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P50
description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Michael E Cahill
@ast
Michael E Cahill
@en
Michael E Cahill
@es
Michael E Cahill
@nl
type
label
Michael E Cahill
@ast
Michael E Cahill
@en
Michael E Cahill
@es
Michael E Cahill
@nl
prefLabel
Michael E Cahill
@ast
Michael E Cahill
@en
Michael E Cahill
@es
Michael E Cahill
@nl
P106
P1153
8745687500
P21
P31
P496
0000-0003-0574-2655