about
Recombination-mediated escape from primary CD8+ T cells in acute HIV-1 infection.Differences in HIV-specific T cell responses between HIV-exposed and -unexposed HIV-seronegative individualsThe upstream sequence element of the C2 complement poly(A) signal activates mRNA 3' end formation by two distinct mechanisms.Vertical T cell immunodominance and epitope entropy determine HIV-1 escape.Broadly targeted CD8⁺ T cell responses restricted by major histocompatibility complex E.Recruitment of a basal polyadenylation factor by the upstream sequence element of the human lamin B2 polyadenylation signal.Transcription and polyadenylation in a short human intergenic region.Efficient use of a 'dead-end' GA 5' splice site in the human fibroblast growth factor receptor genes.A novel CD45 transcript involving the alternative splicing of a new exon is widely distributed in lymphoid tissues.The Role of MHC-E in T Cell Immunity Is Conserved among Humans, Rhesus Macaques, and Cynomolgus Macaques.OA06-04. The role of early T-cell responses in subjects with acute HIV-1 infection.The HIV-1 protective -35SNP effect in Caucasians is CD8 T cell mediated.Pathogen-derived HLA-E bound epitopes reveal broad primary anchor pocket tolerability and conformationally malleable peptide bindingAuthor Correction: Pathogen-derived HLA-E bound epitopes reveal broad primary anchor pocket tolerability and conformationally malleable peptide bindingP08-05. Does increased expression of HLA-C allow better control of HIV-1 viral load?HLA-E: exploiting pathogen-host interactions for vaccine development
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Simon Brackenridge
@ast
Simon Brackenridge
@en
Simon Brackenridge
@es
Simon Brackenridge
@nl
type
label
Simon Brackenridge
@ast
Simon Brackenridge
@en
Simon Brackenridge
@es
Simon Brackenridge
@nl
prefLabel
Simon Brackenridge
@ast
Simon Brackenridge
@en
Simon Brackenridge
@es
Simon Brackenridge
@nl
P106
P31
P496
0000-0002-0587-7560