about
BDNF rescues BAF53b-dependent synaptic plasticity and cocaine-associated memory in the nucleus accumbensβ-Amyloid impairs axonal BDNF retrograde trafficking.Eliminating microglia in Alzheimer's mice prevents neuronal loss without modulating amyloid-β pathology.Humanized monoclonal antibody armanezumab specific to N-terminus of pathological tau: characterization and therapeutic potencyNeural stem cells genetically-modified to express neprilysin reduce pathology in Alzheimer transgenic modelsInhibition of soluble TNF signaling in a mouse model of Alzheimer's disease prevents pre-plaque amyloid-associated neuropathology.An update on estrogen: higher cognitive function, receptor mapping, neurotrophic effects.A microfluidic culture platform for CNS axonal injury, regeneration and transport.Caspase-mediated degeneration in Alzheimer's diseaseSwedish Alzheimer mutation induces mitochondrial dysfunction mediated by HSP60 mislocalization of amyloid precursor protein (APP) and beta-amyloid.Integrated analysis of genetic, behavioral, and biochemical data implicates neural stem cell-induced changes in immunity, neurotransmission and mitochondrial function in Dementia with Lewy Body mice.Human neural stem cells improve cognition and promote synaptic growth in two complementary transgenic models of Alzheimer's disease and neuronal lossThe adaptive immune system restrains Alzheimer's disease pathogenesis by modulating microglial function.Increased tauopathy drives microglia-mediated clearance of beta-amyloid.Neuronal-specific overexpression of a mutant valosin-containing protein associated with IBMPFD promotes aberrant ubiquitin and TDP-43 accumulation and cognitive dysfunction in transgenic mice.Abeta inhibits the proteasome and enhances amyloid and tau accumulationNeural stem cells improve cognition via BDNF in a transgenic model of Alzheimer disease.HuCNS-SC Human NSCs Fail to Differentiate, Form Ectopic Clusters, and Provide No Cognitive Benefits in a Transgenic Model of Alzheimer's Disease.Concise review: Can stem cells be used to treat or model Alzheimer's disease?MultiTEP platform-based DNA epitope vaccine targeting N-terminus of tau induces strong immune responses and reduces tau pathology in THY-Tau22 mice.Neural stem cell therapy for neurodegenerative disorders: The role of neurotrophic support.Soluble amyloid precursor protein induces rapid neural differentiation of human embryonic stem cells.Neural Stem Cells Rescue Cognitive and Motor Dysfunction in a Transgenic Model of Dementia with Lewy Bodies through a BDNF-Dependent Mechanism.Response to StemCells Inc.Estrogen receptor-beta colocalizes extensively with parvalbumin-labeled inhibitory neurons in the cortex, amygdala, basal forebrain, and hippocampal formation of intact and ovariectomized adult rats.Human Neural Progenitor Transplantation Rescues Behavior and Reduces α-Synuclein in a Transgenic Model of Dementia with Lewy Bodies.Neural stem cells improve memory in an inducible mouse model of neuronal loss.Anatomical evidence for transsynaptic influences of estrogen on brain-derived neurotrophic factor expression.iPSC-Derived Human Microglia-like Cells to Study Neurological Diseases.Activation of cell cycle proteins in transgenic mice in response to neuronal loss but not amyloid-beta and tau pathology.Longitudinal behavioral and 6-[18F]fluoro-L-DOPA-PET assessment in MPTP-hemiparkinsonian monkeys.Estradiol-induced modulation of estrogen receptor-beta and GABA within the adult neocortex: a potential transsynaptic mechanism for estrogen modulation of BDNF.Gamma oscillations in the entorhinal-hippocampal circuit underlying memory and dementia.Caspase-cleavage of tau is an early event in Alzheimer disease tangle pathologyDevelopment and validation of a simplified method to generate human microglia from pluripotent stem cellsInduction of Mesoderm and Neural Crest-Derived Pericytes from Human Pluripotent Stem Cells to Study Blood-Brain Barrier InteractionsCD11a expression distinguishes infiltrating myeloid cells from plaque-associated microglia in Alzheimer's diseaseMicroglia in Alzheimer's Disease: Exploring How Genetics and Phenotype Influence RiskDeletion of a Csf1r enhancer selectively impacts CSF1R expression and development of tissue macrophage populations
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P50
description
researcher
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wetenschapper
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հետազոտող
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name
Mathew Blurton-Jones
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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type
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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Mathew Blurton-Jones
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P106
P1153
7801531175
P21
P31
P496
0000-0002-7770-7157