about
A Chronic Longitudinal Characterization of Neurobehavioral and Neuropathological Cognitive Impairment in a Mouse Model of Gulf War Agent Exposure.Gulf War agent exposure causes impairment of long-term memory formation and neuropathological changes in a mouse model of Gulf War IllnessAmelioration of experimental autoimmune encephalomyelitis by anatabineChronic Anatabine Treatment Reduces Alzheimer's Disease (AD)-Like Pathology and Improves Socio-Behavioral Deficits in a Transgenic Mouse Model of ADPhospholipid profiling of plasma from GW veterans and rodent models to identify potential biomarkers of Gulf War Illness.The spleen tyrosine kinase (Syk) regulates Alzheimer amyloid-β production and Tau hyperphosphorylation.Feasibility of Predicting MCI/AD Using Neuropsychological Tests and Serum β-AmyloidBrain-derived neurotropic factor polymorphisms, traumatic stress, mild traumatic brain injury, and combat exposure contribute to postdeployment traumatic stressPlasma cytokine IL-6 levels and subjective cognitive decline: preliminary findings.Complementary proteomic approaches reveal mitochondrial dysfunction, immune and inflammatory dysregulation in a mouse model of Gulf War Illness.Anatabine lowers Alzheimer's Aβ production in vitro and in vivo.Selective antihypertensive dihydropyridines lower Aβ accumulation by targeting both the production and the clearance of Aβ across the blood-brain barrier.Reduction of beta-amyloid pathology by celastrol in a transgenic mouse model of Alzheimer's disease.CD40/CD40L interaction induces Abeta production and increases gamma-secretase activity independently of tumor necrosis factor receptor associated factor (TRAF) signaling.CD40 ligation mediates plaque-associated tau phosphorylation in beta-amyloid overproducing mice.Correction: Chronic Anatabine Treatment Reduces Alzheimer's Disease (AD)-Like Pathology and Improves Socio-Behavioral Deficits in a Transgenic Mouse Model of ADModel of Alzheimer's disease amyloid-beta peptide based on a RNA binding protein.Serum beta-amyloid correlates with neuropsychological impairment.Genetics and Other Risk Factors for Past Concussions in Active-Duty Soldiers.Chronic elevation of phosphocholine containing lipids in mice exposed to Gulf War agents pyridostigmine bromide and permethrin.Anti-inflammatory activity of anatabine via inhibition of STAT3 phosphorylation.Plasma Lipidomic Profiling in a Military Population of Mild Traumatic Brain Injury and Post-Traumatic Stress Disorder with Apolipoprotein E ɛ4-Dependent Effect.Anti-angiogenic activity of the mutant Dutch A(beta) peptide on human brain microvascular endothelial cells.Lifelong behavioral and neuropathological consequences of repetitive mild traumatic brain injury.Inhibition of Abeta production by NF-kappaB inhibitors.Proteomic CNS profile of delayed cognitive impairment in mice exposed to Gulf War agents.The granulocyte macrophage colony stimulating factor (GM-CSF) regulates amyloid beta (Abeta) production.Confirmation of association between D10S583 and Alzheimer's disease in a case--control sample.Genomic regulation after CD40 stimulation in microglia: Relevance to Alzheimer's diseaseA high-resolution map of 1.6 Mb in the Down syndrome region: a new map between D21S55 and ETS2A fast, miniaturised in-vitro assay developed for quantification of lipase enzyme activityTranslational potential of long-term decreases in mitochondrial lipids in a mouse model of Gulf War Illness
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description
onderzoeker
@nl
researcher
@en
հետազոտող
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name
Ghania Ait-Ghezala
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Ghania Ait-Ghezala
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Ghania Ait-Ghezala
@es
Ghania Ait-Ghezala
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type
label
Ghania Ait-Ghezala
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Ghania Ait-Ghezala
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Ghania Ait-Ghezala
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Ghania Ait-Ghezala
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Ghania Ait-Ghezala
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Ghania Ait-Ghezala
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Ghania Ait-Ghezala
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Ghania Ait-Ghezala
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P106
P1153
6507554321
P31
P496
0000-0002-9836-8617