about
Pharmacological rescue of mitochondrial deficits in iPSC-derived neural cells from patients with familial Parkinson's disease.Tau enhances α-synuclein aggregation and toxicity in cellular models of synucleinopathy.Upregulated function of mitochondria-associated ER membranes in Alzheimer diseaseDistinct patterns of APP processing in the CNS in autosomal-dominant and sporadic Alzheimer diseaseMild cholesterol depletion reduces amyloid-beta production by impairing APP trafficking to the cell surface.Autosomal-dominant Alzheimer's disease mutations at the same codon of amyloid precursor protein differentially alter Aβ production.Modification of γ-secretase by nitrosative stress links neuronal ageing to sporadic Alzheimer's disease.Triflusal reduces dense-core plaque load, associated axonal alterations and inflammatory changes, and rescues cognition in a transgenic mouse model of Alzheimer's disease.Clinical, neuropathologic, and biochemical profile of the amyloid precursor protein I716F mutation.Prognostic value of plasma β-amyloid levels in patients with acute intracerebral hemorrhage.A novel GSK-3beta inhibitor reduces Alzheimer's pathology and rescues neuronal loss in vivo.Increased localization of APP-C99 in mitochondria-associated ER membranes causes mitochondrial dysfunction in Alzheimer disease.beta-Amyloid disrupts activity-dependent gene transcription required for memory through the CREB coactivator CRTC1.A deeper look at mitochondrial dynamics in Parkinson’s diseasePINK1 Content in Mitochondria is Regulated by ER-Associated Degradation
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P50
description
hulumtuese
@sq
researcher
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wetenschapper
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հետազոտող
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name
Cristina Guardia-Laguarta
@ast
Cristina Guardia-Laguarta
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Cristina Guardia-Laguarta
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Cristina Guardia-Laguarta
@nl
type
label
Cristina Guardia-Laguarta
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Cristina Guardia-Laguarta
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Cristina Guardia-Laguarta
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Cristina Guardia-Laguarta
@nl
prefLabel
Cristina Guardia-Laguarta
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Cristina Guardia-Laguarta
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Cristina Guardia-Laguarta
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Cristina Guardia-Laguarta
@nl
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0000-0003-0811-9068