about
Sorting nexin 6 enhances lamin a synthesis and incorporation into the nuclear envelopeMiro-1 links mitochondria and microtubule Dynein motors to control lymphocyte migration and polarity.CD81 controls sustained T cell activation signaling and defines the maturation stages of cognate immunological synapses.Nuclear envelope lamin-A couples actin dynamics with immunological synapse architecture and T cell activation.Synthesis, transport and incorporation into the nuclear envelope of A-type lamins and inner nuclear membrane proteins.Fast regulation of AP-1 activity through interaction of lamin A/C, ERK1/2, and c-Fos at the nuclear envelopeRole of A-type lamins in signaling, transcription, and chromatin organization.A-type lamins and Hutchinson-Gilford progeria syndrome: pathogenesis and therapy.Nuclear envelope lamin-A as a coordinator of T cell activation.Tumor suppressor p27(Kip1) undergoes endolysosomal degradation through its interaction with sorting nexin 6.ERK1/2 MAP kinases promote cell cycle entry by rapid, kinase-independent disruption of retinoblastoma-lamin A complexes.A mechanism of AP-1 suppression through interaction of c-Fos with lamin A/C.Tetraspanins CD9 and CD151 at the immune synapse support T-cell integrin signaling.Direct demonstration of beta1- and evidence against beta2- and beta3-adrenoceptors, in smooth muscle cells of rat small mesenteric arteries.High-resolution imaging of intravascular atherogenic inflammation in live mice.Conventional CD4+ T cells present bacterial antigens to induce cytotoxic and memory CD8+ T cell responses.Lamin A/C augments Th1 differentiation and response against vaccinia virus and Leishmania major.β2-Adrenoceptor signaling in airway epithelial cells promotes eosinophilic inflammation, mucous metaplasia, and airway contractility.Author Correction: Conventional CD4+ T cells present bacterial antigens to induce cytotoxic and memory CD8+ T cell responses.CD9 Regulates Major Histocompatibility Complex Class II Trafficking in Monocyte-Derived Dendritic Cells.In Vitro Differentiation of Naïve CD4⁺ T Cells: A Tool for Understanding the Development of Atherosclerosis.Increased p53 gene dosage reduces neointimal thickening induced by mechanical injury but has no effect on native atherosclerosisAn In Vivo Mouse Model to Measure Naïve CD4 T Cell Activation, Proliferation and Th1 Differentiation Induced by Bone Marrow-derived Dendritic CellsDisruption of the CCL1-CCR8 axis inhibits vascular Treg recruitment and function and promotes atherosclerosis in miceLamin A/C deficiency in CD4+ T-cells enhances regulatory T-cells and prevents inflammatory bowel disease
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description
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Jose M Gonzalez-Granado
@ast
Jose M Gonzalez-Granado
@en
Jose M Gonzalez-Granado
@es
Jose M Gonzalez-Granado
@nl
type
label
Jose M Gonzalez-Granado
@ast
Jose M Gonzalez-Granado
@en
Jose M Gonzalez-Granado
@es
Jose M Gonzalez-Granado
@nl
prefLabel
Jose M Gonzalez-Granado
@ast
Jose M Gonzalez-Granado
@en
Jose M Gonzalez-Granado
@es
Jose M Gonzalez-Granado
@nl
P106
P1153
55607375400
P21
P31
P496
0000-0002-1177-869X