about
Alpha-B-crystallin induces an immune-regulatory and antiviral microglial response in preactive multiple sclerosis lesionsGlia Open Access Database (GOAD): A comprehensive gene expression encyclopedia of glia cells in health and disease.Increased White Matter Inflammation in Aging- and Alzheimer's Disease BrainGlioma-associated microglia/macrophages display an expression profile different from M1 and M2 polarization and highly express Gpnmb and Spp1.Induction of a common microglia gene expression signature by aging and neurodegenerative conditions: a co-expression meta-analysis.Immune hyperreactivity of Aβ plaque-associated microglia in Alzheimer's disease.Lymphocryptovirus Infection of Nonhuman Primate B Cells Converts Destructive into Productive Processing of the Pathogenic CD8 T Cell Epitope in Myelin Oligodendrocyte GlycoproteinNext generation transcriptomics and genomics elucidate biological complexity of microglia in health and disease.Transcriptional control of microglia phenotypes in health and disease.Identification of highly connected hub genes in the protective response program of human macrophages and microglia activated by alpha B-crystallin.Activation of an immune-regulatory macrophage response and inhibition of lung inflammation in a mouse model of COPD using heat-shock protein alpha B-crystallin-loaded PLGA microparticles.Identification of a conserved and acute neurodegeneration-specific microglial transcriptome in the zebrafish.CD14 is a key organizer of microglial responses to CNS infection and injury.Enhanced microglial pro-inflammatory response to lipopolysaccharide correlates with brain infiltration and blood-brain barrier dysregulation in a mouse model of telomere shortening.An environment-dependent transcriptional network specifies human microglia identity.Demyelination during multiple sclerosis is associated with combined activation of microglia/macrophages by IFN-γ and alpha B-crystallin.A novel microglial subset plays a key role in myelinogenesis in developing brain.Priming of microglia in a DNA-repair deficient model of accelerated aging.Transcriptomic analysis of purified human cortical microglia reveals age-associated changes.Aging, microglia and cytoskeletal regulation are key factors in the pathological evolution of the APP23 mouse model for Alzheimer's disease.Analysis of Genetically Diverse Macrophages Reveals Local and Domain-wide Mechanisms that Control Transcription Factor Binding and Function.Brain cell type-specific enhancer-promoter interactome maps and disease-risk association
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description
researcher
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wetenschapper
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հետազոտող
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name
Inge Holtman
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Inge R Holtman
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Inge R Holtman
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type
label
Inge Holtman
@nl
Inge R Holtman
@en
Inge R Holtman
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altLabel
I. Holtman
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I.R. Holtman
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Inge R Holtman
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Inge R. Holtman
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prefLabel
Inge Holtman
@nl
Inge R Holtman
@en
Inge R Holtman
@es
P106
P31
P496
0000-0001-9752-4756
P7449
PRS1319863