about
Vestigial is required during late-stage muscle differentiation in Drosophila melanogaster embryos.Functional characterization of the Drosophila MRP (mitochondrial RNA processing) RNA geneMolecular and functional analysis of scalloped recessive lethal alleles in Drosophila melanogasterA stem-loop structure in the wingless transcript defines a consensus motif for apical RNA transport.Alternative requirements for Vestigial, Scalloped, and Dmef2 during muscle differentiation in Drosophila melanogaster.A Drosophila model for the Zellweger spectrum of peroxisome biogenesis disorders.The chemotherapeutic agent paclitaxel inhibits autophagy through two distinct mechanisms that regulate apoptosis.Peroxisome-Mediated Metabolism Is Required for Immune Response to Microbial Infection.Loss of the Drosophila melanogaster DEAD box protein Ddx1 leads to reduced size and aberrant gametogenesis.A multiprotein complex that mediates translational enhancement in Drosophila.Moesin is involved in polarity maintenance and cortical remodelling during asymmetric cell division.The activity of the Drosophila Vestigial protein is modified by Scalloped-dependent phosphorylation.RNA interference effector proteins localize to mobile cytoplasmic puncta in Schizosaccharomyces pombe.The effect of dominant vestigial alleles upon vestigial-mediated wing patterning during development of Drosophila melanogaster.Distinguishable functions for engrailed and invected in anterior-posterior patterning in the Drosophila wing.Apical localization of wingless transcripts is required for wingless signaling.Peroxisome Protein Prediction in Drosophila melanogasterDistinct Roles for Peroxisomal Targeting Signal Receptors Pex5 and Pex7 inA Systematic Cell-Based Analysis of Localization of Predicted Drosophila Peroxisomal ProteinsPeroxisome-Mediated Metabolism Is Required for Immune Response to Microbial InfectionDysfunctional peroxisomes compromise gut structure and host defense by increased cell death and Tor-dependent autophagyImpaired peroxisomal import in Drosophila oenocytes causes cardiac dysfunction by inducing upd3 as a peroxikine
P50
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Andrew J Simmonds
@ast
Andrew J Simmonds
@en
Andrew J Simmonds
@es
Andrew J Simmonds
@nl
type
label
Andrew J Simmonds
@ast
Andrew J Simmonds
@en
Andrew J Simmonds
@es
Andrew J Simmonds
@nl
prefLabel
Andrew J Simmonds
@ast
Andrew J Simmonds
@en
Andrew J Simmonds
@es
Andrew J Simmonds
@nl
P106
P31
P496
0000-0001-7165-9302