about
Identification of the cis-acting endoplasmic reticulum stress response element responsible for transcriptional induction of mammalian glucose-regulated proteins. Involvement of basic leucine zipper transcription factorsIdentification of the G13 (cAMP-response-element-binding protein-related protein) gene product related to activating transcription factor 6 as a transcriptional activator of the mammalian unfolded protein responseComplementary signaling pathways regulate the unfolded protein response and are required for C. elegans developmentDerlin-2 and Derlin-3 are regulated by the mammalian unfolded protein response and are required for ER-associated degradationpXBP1(U) encoded in XBP1 pre-mRNA negatively regulates unfolded protein response activator pXBP1(S) in mammalian ER stress responseUnconventional splicing of XBP1 mRNA occurs in the cytoplasm during the mammalian unfolded protein responseEndoplasmic reticulum stress-induced formation of transcription factor complex ERSF including NF-Y (CBF) and activating transcription factors 6alpha and 6beta that activates the mammalian unfolded protein responseA serine protease inhibitor prevents endoplasmic reticulum stress-induced cleavage but not transport of the membrane-bound transcription factor ATF6.Activation of mammalian unfolded protein response is compatible with the quality control system operating in the endoplasmic reticulumXBP1 is critical to protect cells from endoplasmic reticulum stress: evidence from Site-2 protease-deficient Chinese hamster ovary cells.ER stress and diseases.ER stress response, peroxisome proliferation, mitochondrial unfolded protein response and Golgi stress response.The essential biology of the endoplasmic reticulum stress response for structural and computational biologists.TFE3 is a bHLH-ZIP-type transcription factor that regulates the mammalian Golgi stress response.Organelle autoregulation-stress responses in the ER, Golgi, mitochondria and lysosome.Endoplasmic reticulum stress in kidney function and disease.TFE3, HSP47, and CREB3 Pathways of the Mammalian Golgi Stress Response.Endoplasmic Reticulum (ER) Stress and Endocrine Disorders.Endoplasmic reticulum stress and apoptosis contribute to the pathogenesis of dominantly inherited isolated GH deficiency due to GH1 gene splice site mutations.pXBP1(U), a negative regulator of the unfolded protein response activator pXBP1(S), targets ATF6 but not ATF4 in proteasome-mediated degradation.dutA RNA functions as an untranslatable RNA in the development of Dictyostelium discoideum.XBP1 is essential for survival under hypoxic conditions and is required for tumor growth.The endoplasmic reticulum stress response is stimulated through the continuous activation of transcription factors ATF6 and XBP1 in Ins2+/Akita pancreatic beta cells.The fusion oncoprotein PML-RARalpha induces endoplasmic reticulum (ER)-associated degradation of N-CoR and ER stress.Cloning and characterization of the mitochondrial HSP60-encoding gene of Schizosaccharomyces pombe.Reduction of disulfide bridges in the lumenal domain of ATF6 in response to glucose starvation.MLX Is a Transcriptional Repressor of the Mammalian Golgi Stress Response.Analysis of ATF6 activation in Site-2 protease-deficient Chinese hamster ovary cells.RNA polymerase II transcribes Dictyostelium untranslatable gene, dutA, specifically in the developmental phase.Expression of Dictyostelium early gene, dutA, is independent of cAMP pulses but dependent on protein kinase AA time-dependent phase shift in the mammalian unfolded protein response[Multiphase defense system in mammalian unfolded protein response][Molecular biology of the ER stress response]ATF6 is a transcription factor specializing in the regulation of quality control proteins in the endoplasmic reticulumTranscriptional induction of mammalian ER quality control proteins is mediated by single or combined action of ATF6alpha and XBP1Differential contributions of ATF6 and XBP1 to the activation of endoplasmic reticulum stress-responsive cis-acting elements ERSE, UPRE and ERSE-IINovel cis-acting element GASE regulates transcriptional induction by the Golgi stress responseUltraviolet a induces endoplasmic reticulum stress response in human dermal fibroblasts[Cytoplasmic mRNA splicing]UBC9 regulates the stability of XBP1, a key transcription factor controlling the ER stress response
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description
onderzoeker
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researcher
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հետազոտող
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H Yoshida
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H Yoshida
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H Yoshida
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H Yoshida
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H Yoshida
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H Yoshida
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H Yoshida
@es
H Yoshida
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H Yoshida
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H Yoshida
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H Yoshida
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H Yoshida
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P106
P1153
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7408718562
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0000-0002-5773-1408