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Skeletal Muscle Insulin Resistance and Absence of Inflammation Characterize Insulin-Resistant Grade I Obese Women.Muscle Contraction Induces Acute Hydroxymethylation of the Exercise-Responsive Gene Nr4a3.Defects in TLR3 expression and RNase L activation lead to decreased MnSOD expression and insulin resistance in muscle cells of obese people.Ionizing Radiation Potentiates High-Fat Diet-Induced Insulin Resistance and Reprograms Skeletal Muscle and Adipose Progenitor Cells.Evidence Suggesting Absence of Mitochondrial DNA Methylation.Combination of nutritional polyphenols supplementation with exercise training counteracts insulin resistance and improves endurance in high-fat diet-induced obese rats.Endurance training remodels sperm-borne small RNA expression and methylation at neurological gene hotspots.In Situ Fixation Redefines Quiescence and Early Activation of Skeletal Muscle Stem Cells.Decreased RNF41 expression leads to insulin resistance in skeletal muscle of obese women.Preadipocytes from obese humans with type 2 diabetes are epigenetically reprogrammed at genes controlling adipose tissue function.Exercise training alters the genomic response to acute exercise in human adipose tissue.No Additive Effects of Polyphenol Supplementation and Exercise Training on White Adiposity Determinants of High-Fat Diet-Induced Obese Insulin-Resistant Rats.T cell epigenetic remodeling and accelerated epigenetic aging are linked to long-term immune alterations in childhood cancer survivorsMuscle mitochondrial metabolism and calcium signaling impairment in patients treated with statins
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description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Odile Fabre
@ast
Odile Fabre
@en
Odile Fabre
@es
Odile Fabre
@nl
type
label
Odile Fabre
@ast
Odile Fabre
@en
Odile Fabre
@es
Odile Fabre
@nl
prefLabel
Odile Fabre
@ast
Odile Fabre
@en
Odile Fabre
@es
Odile Fabre
@nl
P106
P1153
56641320400
P31
P496
0000-0003-0892-8412