about
CD39: a regulatory role in childhood arthritisGenome-wide data reveal novel genes for methotrexate response in a large cohort of juvenile idiopathic arthritis casesGeneration of novel pharmacogenomic candidates in response to methotrexate in juvenile idiopathic arthritis: correlation between gene expression and genotype.Association of the 5-aminoimidazole-4-carboxamide ribonucleotide transformylase gene with response to methotrexate in juvenile idiopathic arthritis.Genetics of juvenile idiopathic arthritis: new tools bring new approaches.Th1 and Th17 cell subpopulations are enriched in the peripheral blood of patients with systemic juvenile idiopathic arthritisInterleukin-17-producing T cells are enriched in the joints of children with arthritis, but have a reciprocal relationship to regulatory T cell numbers.Correlation of low CD73 expression on synovial lymphocytes with reduced adenosine generation and higher disease severity in juvenile idiopathic arthritis.Overexpression of MHC class I heavy chain protein in young skeletal muscle leads to severe myositis: implications for juvenile myositisThe Toll-like receptor 4 agonist MRP8/14 protein complex is a sensitive indicator for disease activity and predicts relapses in systemic-onset juvenile idiopathic arthritis.CD25+ CD4+ T cells compete with naive CD4+ T cells for IL-2 and exploit it for the induction of IL-10 production.Modulation of dendritic cell function by naive and regulatory CD4+ T cells.Methotrexate in childhood arthritis: effects on gene expression.The transcription factor crem-αlpha regulates inflammatory T cell subsets in juvenile idiopathic arthritis.A subgroup of juvenile idiopathic arthritis patients who respond well to methotrexate are identified by the serum biomarker MRP8/14 protein.S100A12 Is Associated with Response to Therapy in Juvenile Idiopathic Arthritis.The influence of CD4 T-cell subsets on control of CD4 T-cell-mediated graft-versus-host disease.Transcriptional profiles of JIA patient blood with subsequent poor response to methotrexate.Characterising inflammatory markers in two childhood autoimmune diseases (JIA and JDM) pre and post methotrexate.Can inflammatory markers predict response to methotrexate in JIA? Results from the CHARM study.The transcription factor CREM drives an inflammatory phenotype of T cells in oligoarticular juvenile idiopathic arthritis.PReS-FINAL-1001: Lymphocytes from the inflamed joint of juvenile idiopathic arthritis patients express reduced levels of cd73 and have a functional defect in adenosine production.Identification of enhanced IFN-γ signaling in polyarticular juvenile idiopathic arthritis with mass cytometry
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description
onderzoeker
@nl
researcher ORCID ID = 0000-0001-9744-4879
@en
name
Halima Moncrieffe
@ast
Halima Moncrieffe
@en
Halima Moncrieffe
@es
Halima Moncrieffe
@nl
type
label
Halima Moncrieffe
@ast
Halima Moncrieffe
@en
Halima Moncrieffe
@es
Halima Moncrieffe
@nl
prefLabel
Halima Moncrieffe
@ast
Halima Moncrieffe
@en
Halima Moncrieffe
@es
Halima Moncrieffe
@nl
P106
P1153
8323788900
P31
P496
0000-0001-9744-4879