about
The novel MMS-inducible gene Mif1/KIAA0025 is a target of the unfolded protein response pathwaycDNA micro array identification of a gene differentially expressed in adenovirus type 5- versus type 12-transformed cellsSuppression of anoikis and induction of metastasis by the neurotrophic receptor TrkBExpression of BNIP3 in invasive breast cancer: correlations with the hypoxic response and clinicopathological featuresInvincible DNA tethers: covalent DNA anchoring for enhanced temporal and force stability in magnetic tweezers experiments.Genetic depletion and pharmacological targeting of αv integrin in breast cancer cells impairs metastasis in zebrafish and mouse xenograft models.Spheroid assay to measure TGF-β-induced invasion.APP and APLP1 are degraded through autophagy in response to proteasome inhibition in neuronal cells.Strand separation establishes a sustained lock at the Tus-Ter replication fork barrier.Essential validation methods for E. coli strains created by chromosome engineeringInduction of the SAPK activator MIG-6 by the alkylating agent methyl methanesulfonate.High-throughput, high-force probing of DNA-protein interactions with magnetic tweezers.Mutations of the PU.1 Ets domain are specifically associated with murine radiation-induced, but not human therapy-related, acute myeloid leukaemia.VEGF and inhibitors of TGFbeta type-I receptor kinase synergistically promote blood-vessel formation by inducing alpha5-integrin expression.The APP intracellular domain (AICD) inhibits Wnt signalling and promotes neurite outgrowth.BMP-7 inhibits TGF-β-induced invasion of breast cancer cells through inhibition of integrin β(3) expression.Wnt/β-catenin signal pathway stabilizes APP intracellular domain (AICD) and promotes its transcriptional activity
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researcher ORCID ID = 0000-0002-0990-6234
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Theo van Laar
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0000-0002-0990-6234