about
From pathways to targets: understanding the mechanisms behind polyglutamine disease.A combinatorial approach to identify calpain cleavage sites in the Machado-Joseph disease protein ataxin-3.The calpain-suppressing effects of olesoxime in Huntington's disease.Reduced cell size, chromosomal aberration and altered proliferation rates are characteristics and confounding factors in the STHdh cell model of Huntington disease.Calpastatin ablation aggravates the molecular phenotype in cell and animal models of Huntington disease.Karyopherin α-3 is a key protein in the pathogenesis of spinocerebellar ataxia type 3 controlling the nuclear localization of ataxin-3.Environment-dependent striatal gene expression in the BACHD rat model for Huntington disease.Mitochondrial Morphology, Function and Homeostasis Are Impaired by Expression of an N-terminal Calpain Cleavage Fragment of Ataxin-3Vulnerability of frontal brain neurons for the toxicity of expanded ataxin-3Killing Two Angry Birds with One Stone: Autophagy Activation by Inhibiting Calpains in Neurodegenerative Diseases and BeyondGeneration of an induced pluripotent stem cell line from a patient with spinocerebellar ataxia type 3 (SCA3): HIHCNi002-AOlesoxime in neurodegenerative diseases: Scrutinising a promising drug candidateCalpain-1 ablation partially rescues disease-associated hallmarks in models of Machado-Joseph disease
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description
German biologist
@en
German biologist
@en-ca
German biologist
@en-gb
bioloog
@nl
bitheolaí Gearmánach
@ga
biólogo alemán
@es
deutscher Biologe
@de
عالم أحياء ألماني
@ar
name
Jonasz J Weber
@es
Jonasz J Weber
@nl
Jonasz J. Weber
@de
Jonasz J. Weber
@en
type
label
Jonasz J Weber
@es
Jonasz J Weber
@nl
Jonasz J. Weber
@de
Jonasz J. Weber
@en
altLabel
Jonasz Jeremiasz Weber
@de
Jonasz Jeremiasz Weber
@en
prefLabel
Jonasz J Weber
@es
Jonasz J Weber
@nl
Jonasz J. Weber
@de
Jonasz J. Weber
@en
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