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Nuclear factor erythroid 2-related factor 2 facilitates neuronal glutathione synthesis by upregulating neuronal excitatory amino acid transporter 3 expression.The Nrf2-ARE pathway: an indicator and modulator of oxidative stress in neurodegenerationExercise Counteracts Aging-Related Memory Impairment: A Potential Role for the Astrocytic Metabolic ShuttleStem cell transplantation therapy in Parkinson's diseaseRole of Hsp90 in biogenesis of the beta-cell ATP-sensitive potassium channel complexSyntaxin 1A regulates surface expression of beta-cell ATP-sensitive potassium channels.Concerted Trafficking Regulation of Kv2.1 and KATP Channels by Leptin in Pancreatic β-Cells.Nrf2-mediated neuroprotection in the MPTP mouse model of Parkinson's disease: Critical role for the astrocyte.Carbamazepine as a novel small molecule corrector of trafficking-impaired ATP-sensitive potassium channels identified in congenital hyperinsulinism.Leptin regulates KATP channel trafficking in pancreatic β-cells by a signaling mechanism involving AMP-activated protein kinase (AMPK) and cAMP-dependent protein kinase (PKA).Pharmacological rescue of trafficking-impaired ATP-sensitive potassium channels.Compounds that correct F508del-CFTR trafficking can also correct other protein trafficking diseases: an in vitro study using cell lines.Carbamazepine inhibits ATP-sensitive potassium channel activity by disrupting channel response to MgADP.NMDA receptors mediate leptin signaling and regulate potassium channel trafficking in pancreatic β-cells.Defective trafficking of Kv2.1 channels in MPTP-induced nigrostriatal degeneration.Monitoring Changes in the Abundance of Endogenously Expressed ATP-Sensitive Potassium (KATP) Channels in the Plasma Membrane Using Surface Biotinylation.Melatonin prevents the dynamin-related protein 1-dependent mitochondrial fission and oxidative insult in the cortical neurons after 1-methyl-4-phenylpyridinium treatment.The comprehensive electrophysiological study of curcuminoids on delayed-rectifier K+ currents in insulin-secreting cells.Mitochondrial defects arise from nucleoside/nucleotide reverse transcriptase inhibitors in neurons: Potential contribution to HIV-associated neurocognitive disorders.Evidence of Decreased Activity in Intermediate-Conductance Calcium-Activated Potassium Channels During Retinoic Acid-Induced Differentiation in Motor Neuron-Like NSC-34 Cells
P50
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P50
description
onderzoeker
@nl
researcher ORCID ID = 0000-0002-2550-6556
@en
name
Pei-Chun Chen
@ast
Pei-Chun Chen
@en
Pei-Chun Chen
@es
Pei-Chun Chen
@nl
type
label
Pei-Chun Chen
@ast
Pei-Chun Chen
@en
Pei-Chun Chen
@es
Pei-Chun Chen
@nl
prefLabel
Pei-Chun Chen
@ast
Pei-Chun Chen
@en
Pei-Chun Chen
@es
Pei-Chun Chen
@nl
P108
P106
P108
P1153
55490202200
P31
P496
0000-0002-2550-6556