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Identification of keratinocyte growth factor as a target of microRNA-155 in lung fibroblasts: implication in epithelial-mesenchymal interactionsmiR-210 is overexpressed in late stages of lung cancer and mediates mitochondrial alterations associated with modulation of HIF-1 activityTumor suppressor function of miR-483-3p on squamous cell carcinomas due to its pro-apoptotic properties."Seed-Milarity" confers to hsa-miR-210 and hsa-miR-147b similar functional activity.The microRNA-130/301 family controls vasoconstriction in pulmonary hypertensionA YAP/TAZ-miR-130/301 molecular circuit exerts systems-level control of fibrosis in a network of human diseases and physiologic conditionsEpigenetic switch drives the conversion of fibroblasts into proinvasive cancer-associated fibroblastsMiR-210 promotes a hypoxic phenotype and increases radioresistance in human lung cancer cell lines.Vascular stiffness mechanoactivates YAP/TAZ-dependent glutaminolysis to drive pulmonary hypertensionp38MAPK builds a hyaluronan cancer niche to drive lung tumorigenesis.Membrane-bound ICAM-1 contributes to the onset of proinvasive tumor stroma by controlling acto-myosin contractility in carcinoma-associated fibroblasts.MicroRNA target identification: lessons from hypoxamiRs.miR-193b/365a cluster controls progression of epidermal squamous cell carcinoma.Impact of MicroRNAs in the Cellular Response to Hypoxia.CDC25A targeting by miR-483-3p decreases CCND-CDK4/6 assembly and contributes to cell cycle arrest.Matrix Remodeling Promotes Pulmonary Hypertension through Feedback Mechanoactivation of the YAP/TAZ-miR-130/301 Circuit.Systems-level regulation of microRNA networks by miR-130/301 promotes pulmonary hypertension.miR-483-3p controls proliferation in wounded epithelial cells.Tumor-Stroma Mechanics Coordinate Amino Acid Availability to Sustain Tumor Growth and MalignancyFactors Associated with Heritable Pulmonary Arterial Hypertension Exert Convergent Actions on the miR-130/301-Vascular Matrix Feedback LoopUBTD1 is a mechano-regulator controlling cancer aggressivenessMatrix Stiffening and EGFR Cooperate to Promote the Collective Invasion of Cancer CellsLongitudinal Evaluation of Pulmonary Arterial Hypertension in a Rhesus Macaque (Macaca mulatta) Model of HIV InfectionBmpr2 Mutant Rats Develop Pulmonary and Cardiac Characteristics of Pulmonary Arterial HypertensionBOLA (BolA Family Member 3) Deficiency Controls Endothelial Metabolism and Glycine Homeostasis in Pulmonary HypertensionThe molecular rationale for therapeutic targeting of glutamine metabolism in pulmonary hypertensionInhibition of CHK 1 (Checkpoint Kinase 1) Elicits Therapeutic Effects in Pulmonary Arterial HypertensionCharacterization of Kcnk3-Mutated Rat, a Novel Model of Pulmonary HypertensionMechanical forces rewire metabolism in the tumor niche
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description
researcher, ORCID id # 0000-0002-4801-9902
@en
wetenschapper
@nl
name
Thomas Bertero
@ast
Thomas Bertero
@en
Thomas Bertero
@es
Thomas Bertero
@nl
type
label
Thomas Bertero
@ast
Thomas Bertero
@en
Thomas Bertero
@es
Thomas Bertero
@nl
prefLabel
Thomas Bertero
@ast
Thomas Bertero
@en
Thomas Bertero
@es
Thomas Bertero
@nl
P106
P31
P496
0000-0002-4801-9902