about
Bmi-1 promotes neural stem cell self-renewal and neural development but not mouse growth and survival by repressing the p16Ink4a and p19Arf senescence pathways.Increasing p16INK4a expression decreases forebrain progenitors and neurogenesis during ageingEnteric glia are multipotent in culture but primarily form glia in the adult rodent gut.Sox17 expression confers self-renewal potential and fetal stem cell characteristics upon adult hematopoietic progenitorsStem cell self-renewal and cancer cell proliferation are regulated by common networks that balance the activation of proto-oncogenes and tumor suppressors.Haematopoietic stem cells do not asymmetrically segregate chromosomes or retain BrdU.Mechanisms of stem cell self-renewal.Senescence in Health and Disease.Bmi-1 over-expression in neural stem/progenitor cells increases proliferation and neurogenesis in culture but has little effect on these functions in vivo.Enhanced purification of fetal liver hematopoietic stem cells using SLAM family receptors.Transient CDK4/6 inhibition protects hematopoietic stem cells from chemotherapy-induced exhaustion.
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P50
description
onderzoeker
@nl
researcher, ORCID id # 0000-0001-8842-7137
@en
name
Shenghui He
@ast
Shenghui He
@en
Shenghui He
@nl
type
label
Shenghui He
@ast
Shenghui He
@en
Shenghui He
@nl
prefLabel
Shenghui He
@ast
Shenghui He
@en
Shenghui He
@nl
P108
P106
P108
P31
P496
0000-0001-8842-7137