about
Failure to detect xenotropic murine leukemia virus-related virus in blood of individuals at high risk of blood-borne viral infectionsA contaminant-free assessment of Endogenous Retroviral RNA in human plasmaPhylogenetic analysis consistent with a clinical history of sexual transmission of HIV-1 from a single donor reveals transmission of highly distinct variants.Low copy target detection by Droplet Digital PCR through application of a novel open access bioinformatic pipeline, 'definetherain'High-multiplicity HIV-1 infection and neutralizing antibody evasion mediated by the macrophage-T cell virological synapse.Modelling the evolution and spread of HIV immune escape mutantsShort-course antiretroviral therapy in primary HIV infection.Mutagenically separated PCR assay for rapid detection of M41L and K70R zidovudine resistance mutations in CRF01_AE (subtype E) human immunodeficiency virus type 1.Quantifiable cytotoxic T lymphocyte responses and HLA-related risk of progression to AIDSHIV-1 DNA predicts disease progression and post-treatment virological control.Co-operative additive effects between HLA alleles in control of HIV-1Adaptation of HIV-1 to human leukocyte antigen class IImpact of HLA-driven HIV adaptation on virulence in populations of high HIV seroprevalence.Progression to AIDS in South Africa is associated with both reverting and compensatory viral mutations.HIV transmission. Selection bias at the heterosexual HIV-1 transmission bottleneck.Duration of HIV-1 viral suppression on cessation of antiretroviral therapy in primary infection correlates with time on therapy.Structured observations reveal slow HIV-1 CTL escape.Discordant Impact of HLA on Viral Replicative Capacity and Disease Progression in Pediatric and Adult HIV Infection.Evaluation of Viremia Frequencies of a Novel Human Pegivirus by Using Bioinformatic Screening and PCRExhaustion of Activated CD8 T Cells Predicts Disease Progression in Primary HIV-1 Infection.Histone Deacetylase Inhibitors Enhance CD4 T Cell Susceptibility to NK Cell Killing but Reduce NK Cell Function.Immunological biomarkers predict HIV-1 viral rebound after treatment interruptionHIV control through a single nucleotide on the HLA-B locus.Human endogenous retrovirus (HERV) expression is not induced by treatment with the histone deacetylase (HDAC) inhibitors in cellular models of HIV-1 latencyEarly and nonreversible decrease of CD161++ /MAIT cells in HIV infection.Tenofovir Disoproxil Fumarate Fails to Prevent HIV Acquisition or the Establishment of a Viral Reservoir: Two Case Reports.Impact of pre-adapted HIV transmission.HLA-A*7401-mediated control of HIV viremia is independent of its linkage disequilibrium with HLA-B*5703Towards an HIV cure: science and debate from the International AIDS Society 2013 symposium.Virological Blips and Predictors of Post Treatment Viral Control After Stopping ART Started in Primary HIV Infection.Post-treatment control or treated controllers? Viral remission in treated and untreated primary HIV infection.Differential escape patterns within the dominant HLA-B*57:03-restricted HIV Gag epitope reflect distinct clade-specific functional constraints.New approaches in HIV eradication research.A cure for HIV: is it in sight?Advancing the HIV cure agenda: the next 5 years.HIV-1-specific CD4(+) responses in primary HIV-1 infection predict disease progression.Increased levels of CD4 T-cell activation in individuals with CXCR4 using viruses in primary HIV-1 infection.Short Communication: Lack of Effect of Maraviroc Intensification on Blood and Gut Reservoir.The Role of CD4+ T Follicular Helper Cells in HIV Infection: From the Germinal Center to the PeripheryNonprogressing HIV-infected children share fundamental immunological features of nonpathogenic SIV infection.
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description
researcher, ORCID id # 0000-0001-7163-7277
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wetenschapper
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name
John Frater
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John Frater
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John Frater
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John Frater
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type
label
John Frater
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John Frater
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John Frater
@es
John Frater
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prefLabel
John Frater
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John Frater
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John Frater
@es
John Frater
@nl
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P1153
35573097200
6701540433
P21
P31
P496
0000-0001-7163-7277