Suppression of a mitochondrial point mutation in a tRNA gene can cast light on the mechanisms of 3? end-processing
about
Pathogenic mechanism of a human mitochondrial tRNAPhe mutation associated with myoclonic epilepsy with ragged red fibers syndrome.The yeast counterparts of human 'MELAS' mutations cause mitochondrial dysfunction that can be rescued by overexpression of the mitochondrial translation factor EF-Tu.Suppression of a nuclear frameshift mutation by a mitochondrial tRNA in the yeast Kluyveromyces lactis.
P2860
Suppression of a mitochondrial point mutation in a tRNA gene can cast light on the mechanisms of 3? end-processing
description
article
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в травні 1994
@uk
name
Suppression of a mitochondrial ...... echanisms of 3? end-processing
@en
Suppression of a mitochondrial ...... echanisms of 3? end-processing
@nl
type
label
Suppression of a mitochondrial ...... echanisms of 3? end-processing
@en
Suppression of a mitochondrial ...... echanisms of 3? end-processing
@nl
prefLabel
Suppression of a mitochondrial ...... echanisms of 3? end-processing
@en
Suppression of a mitochondrial ...... echanisms of 3? end-processing
@nl
P50
P356
P1433
P1476
Suppression of a mitochondrial ...... echanisms of 3' end-processing
@en
P2093
P2888
P304
P356
10.1007/BF00351785
P577
1994-05-01T00:00:00Z